PPARγ agonists inhibit cell growth and suppress the expression of cyclin D1 and EGF-like growth factors in ras-transformed rat intestinal epithelial cells

International Journal of Cancer

Volumn 94, Issue 3, 2001, Pages 335-342

  Kitamura, Shinji ^a,b   Miyazaki, Yoshiji ^a   Hiraoka, Shintaro ^a   Nagasawa, Yutaka ^a   Toyota, Miyuki ^a   Takakura, Rena ^a   Kiyohara, Tatsuya ^a   Shinomura, Yasuhisa ^a   Matsuzawa, Yuji ^a  

^a OSAKA UNIVERSITY   (Japan)

^b OSAKA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

Author keywords

AP 1; Ets; Intestinal epithelial cell; PPAR

Indexed keywords

AMPHIREGULIN; COMPLEMENTARY DNA; CYCLIN D1; HEPARIN BINDING EPIDERMAL GROWTH FACTOR; LUCIFERASE; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR AGONIST; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA; RAS PROTEIN; ROSIGLITAZONE; TRANSCRIPTION FACTOR AP 1; TROGLITAZONE; 2,4 THIAZOLIDINEDIONE; 2,4-THIAZOLIDINEDIONE; ANTIDIABETIC AGENT; CELL RECEPTOR; CHROMAN DERIVATIVE; EPIDERMAL GROWTH FACTOR; LIGAND; MESSENGER RNA; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR; PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR; THIAZOLE DERIVATIVE; TRANSCRIPTION FACTOR;

ANIMAL CELL; ARTICLE; CANCER CELL; CANCER INHIBITION; CELL CYCLE G1 PHASE; CELL GROWTH; CELL TRANSFORMATION; CONTROLLED STUDY; ELECTROPHORETIC MOBILITY; ENZYME ASSAY; GENE EXPRESSION; INTESTINE EPITHELIUM CELL; MOLECULAR CLONING; NONHUMAN; PRIORITY JOURNAL; RAT; TRANSCRIPTION REGULATION; ANIMAL; BIOSYNTHESIS; CELL DIVISION; CELL LINE; CELL NUCLEUS; CYTOLOGY; DOSE RESPONSE; DRUG EFFECT; DRUG POTENTIATION; EPITHELIUM CELL; FLOW CYTOMETRY; GENETIC TRANSCRIPTION; GENETIC TRANSFECTION; HUMAN; INTESTINE; METABOLISM; NORTHERN BLOTTING; PLASMID; PROMOTER REGION; TIME; TRANSACTIVATION; WESTERN BLOTTING;

ANIMAL; BLOTTING, NORTHERN; BLOTTING, WESTERN; CELL DIVISION; CELL LINE; CELL LINE, TRANSFORMED; CELL NUCLEUS; CHROMANS; CYCLIN D1; DNA, COMPLEMENTARY; DOSE-RESPONSE RELATIONSHIP, DRUG; EPIDERMAL GROWTH FACTOR; EPITHELIAL CELLS; FLOW CYTOMETRY; HUMAN; HYPOGLYCEMIC AGENTS; INTESTINES; LIGANDS; LUCIFERASE; PLASMIDS; PROMOTER REGIONS (GENETICS); RAS PROTEINS; RATS; RECEPTORS, CYTOPLASMIC AND NUCLEAR; RNA, MESSENGER; SUPPORT, NON-U.S. GOV'T; THIAZOLES; TIME FACTORS; TRANS-ACTIVATION (GENETICS); TRANSCRIPTION FACTOR AP-1; TRANSCRIPTION FACTORS; TRANSCRIPTION, GENETIC; TRANSFECTION;

EID: 0035497756     PISSN: 00207136     EISSN: None     Source Type: Journal    
DOI: 10.1002/ijc.1470     Document Type: Article

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