HIV-1 Tat induces microvascular endothelial apoptosis through caspase activation

Journal of Immunology

Volumn 167, Issue 5, 2001, Pages 2766-2771

  Park, I W ^a   Ullrich, C K ^a   Schoenberger, E ^a   Ganju, R K ^a   Groopman, J E ^a  

^a BETH ISRAEL DEACONESS MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 3; FAS ANTIGEN; FAS LIGAND; PROTEIN BAD; PROTEIN BAX; PROTEIN BCL 2; TRANSACTIVATOR PROTEIN; VIRUS PROTEIN;

APOPTOSIS; ARTICLE; CHROMATIN STRUCTURE; CONTROLLED STUDY; CYTOKINE RELEASE; ENDOTHELIUM CELL; ENZYME ACTIVATION; ENZYME INHIBITION; HUMAN; HUMAN CELL; HUMAN IMMUNODEFICIENCY VIRUS 1; NICK END LABELING; PRIORITY JOURNAL; PROTEIN DEGRADATION; VIRUS TRANSCRIPTION;

EID: 0035451405     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.167.5.2766     Document Type: Article

References (26)

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2. The Tat protein of human immunodeficiency virus type 1, a growth factor for AIDS Kaposi sarcoma and cytokine-activated vascular cells, induces adhesion of the same cell types by using integrin receptors recognizing the RGD amino acid sequence
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4. 5 integrin to the basic domain of the HIV Tat protein and vitronectin
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