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Volumn 30, Issue 1, 2001, Pages 9-21
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Superoxide radical-initiated apoptotic signalling pathway in selenite-treated HepG2 cells: Mitochondria serve as the main target
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Author keywords
Apoptosis; Caspase; Cytochrome c; Free radicals; Mitochondria; Selenite; Superoxide radical
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Indexed keywords
ACETYLCYSTEINE;
CASPASE 3;
CYCLOSPORIN A;
CYTOCHROME C;
DNA FRAGMENT;
GLUTATHIONE;
RADICAL;
SODIUM SELENITE;
SUPEROXIDE;
TEMPOL;
TRIFLUOPERAZINE;
APOPTOSIS;
ARTICLE;
CELL STRAIN HEPG2;
CONTROLLED STUDY;
DNA DAMAGE;
ENZYME ACTIVITY;
ENZYME RELEASE;
HUMAN;
HUMAN CELL;
LIVER MITOCHONDRION;
MEMBRANE DEPOLARIZATION;
MEMBRANE PERMEABILITY;
MITOCHONDRIAL MEMBRANE;
PRIORITY JOURNAL;
REDUCTION;
SIGNAL TRANSDUCTION;
ACETYLCYSTEINE;
APOPTOSIS;
CARCINOMA, HEPATOCELLULAR;
CASPASE 3;
CASPASES;
CYCLIC N-OXIDES;
CYCLOSPORINE;
CYTOCHROME C GROUP;
DNA FRAGMENTATION;
DOSE-RESPONSE RELATIONSHIP, DRUG;
ENZYME ACTIVATION;
ENZYME INHIBITORS;
GLUTATHIONE;
HUMANS;
IN SITU NICK-END LABELING;
KINETICS;
LIVER NEOPLASMS;
MEMBRANE POTENTIALS;
MITOCHONDRIA;
SIGNAL TRANSDUCTION;
SODIUM SELENITE;
SPIN LABELS;
SUPEROXIDES;
TRIFLUOPERAZINE;
TUMOR CELLS, CULTURED;
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EID: 0035185334
PISSN: 08915849
EISSN: None
Source Type: Journal
DOI: 10.1016/S0891-5849(00)00421-4 Document Type: Article |
Times cited : (121)
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References (59)
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