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Volumn 167, Issue 1, 2001, Pages 59-64

G protein and cAMP-dependent protein kinase mediate amyloid β-peptide inhibition of neuronal glucose uptake

Author keywords

Alzheimer's disease; cAMP; G protein; Glucose uptake; Hippocampal neuron; amyloid

Indexed keywords

ADENYLATE CYCLASE; AMYLOID BETA PROTEIN; BUCLADESINE; CHOLERA TOXIN; CYCLIC AMP; CYCLIC AMP DEPENDENT PROTEIN KINASE; FORSKOLIN; GLUCOSE; GUANINE NUCLEOTIDE BINDING PROTEIN; KT 5720;

EID: 0035152374     PISSN: 00144886     EISSN: None     Source Type: Journal    
DOI: 10.1006/exnr.2000.7519     Document Type: Article
Times cited : (23)

References (40)
  • 10
  • 11
    • 0025856239 scopus 로고
    • Predominant abnormality in cerebral glucose utilization in late-onset dementia of the Alzheimer type: A cross-sectional comparison against advanced late-onset and incipient early-onset cases
    • (1991) J. Neural Transm. , vol.3 , pp. 1-14
    • Hoyer, S.1    Nitsch, R.2    Oesterreich, K.3
  • 21
    • 0030779677 scopus 로고    scopus 로고
    • Cellular actions of β-amyloid precursor protein and its soluble and fibrillogenic derivatives
    • (1997) Physiol. Rev. , vol.77 , pp. 1081-1132
    • Mattson, M.1
  • 33
    • 0024709982 scopus 로고
    • Signal sorting and amplification through G protein-coupled receptors
    • (1989) Neuron , vol.3 , pp. 141-152
    • Ross, E.M.1
  • 40
    • 0029896354 scopus 로고    scopus 로고
    • Mechanisms of neuronal degeneration in Alzheimer's disease
    • (1996) Neuron , vol.16 , pp. 921-932
    • Yankner, B.A.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.