Th1/Th2 predominance and proinflammatory cytokines determine the clinicopathological severity of IgA nephropathy

Nephrology Dialysis Transplantation

Volumn 16, Issue 2, 2001, Pages 269-275

  Lim, Chun Soo ^a   Zheng, Shouhuan ^a   Kim, Yon Su ^a   Ahn, Curie ^a   Han, Jin Suk ^a   Kim, Suhnggwon ^a   Lee, Jung Sang ^a   Chae, Dong Wan ^b   Koo, Ja Ryong ^b   Chun, Roh Won ^b   Noh, Jung Woo ^b  

^a Seoul National University College of Medicine   (South Korea)

^b Hallym University College of Medicine   (South Korea)

Author keywords

Cytokine; IgA nephropathy; Interferon ; Interleukin 10; Interleukin 8; Semi quantitative reverse transcriptase polymerase chain reaction

Indexed keywords

CHEMOKINE; CYTOKINE; GAMMA INTERFERON; INTERLEUKIN 10; INTERLEUKIN 15; INTERLEUKIN 1BETA; INTERLEUKIN 2; INTERLEUKIN 6; INTERLEUKIN 8; MONOCYTE CHEMOTACTIC PROTEIN 1; RANTES; TUMOR NECROSIS FACTOR ALPHA;

ADOLESCENT; ADULT; AGED; ARTICLE; CELL INFILTRATION; CELL PROLIFERATION; CLINICAL ARTICLE; CLINICAL FEATURE; CONTROLLED STUDY; DISEASE SEVERITY; FEMALE; GENE EXPRESSION; GENETIC TRANSCRIPTION; GLOMERULONEPHRITIS; GLOMERULOSCLEROSIS; HUMAN; HYPOTHESIS; IMMUNOFLUORESCENCE MICROSCOPY; IMMUNOGLOBULIN A NEPHROPATHY; INTERSTITIAL NEPHRITIS; KIDNEY BIOPSY; KIDNEY FAILURE; KIDNEY FUNCTION; MALE; MESANGIUM; PATHOGENESIS; PRIORITY JOURNAL; PROTEINURIA; QUANTITATIVE ASSAY; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; TH1 CELL; TH2 CELL;

EID: 0035142568     PISSN: 09310509     EISSN: None     Source Type: Journal    
DOI: 10.1093/ndt/16.2.269     Document Type: Article

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