No vascular control in Duchenne muscular dystrophy

Nature Medicine

Volumn 7, Issue 1, 2001, Pages 27-29

  Crosbie, R H ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DYSTROPHIN; NITRIC OXIDE SYNTHASE;

CLINICAL FEATURE; DUCHENNE MUSCULAR DYSTROPHY; GENE MUTATION; HEART FAILURE; HUMAN; MUSCLE ATROPHY; MUSCLE NECROSIS; MUSCLE REGENERATION; MUSCLE WEAKNESS; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; RESPIRATORY FAILURE; SHORT SURVEY; VASOCONSTRICTION; VASOMOTOR REFLEX;

HUMANS; MUSCLE, SKELETAL; MUSCULAR DYSTROPHY, DUCHENNE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE TYPE I; VASOCONSTRICTION;

EID: 0035138623     PISSN: 10788956     EISSN: None     Source Type: Journal    
DOI: 10.1038/83309     Document Type: Short Survey

References (18)

1. Functional muscle ischemia in neuronal nitric oxide synthase-deficient skeletal muscle of children with Duchenne muscular dystrophy
2. Association of dystrophin and an integral membrane glycoprotein
3. Glycoprotein complex anchoring dystrophin to sarcolemma
4. Molecular basis of muscular dystrophies
5. Deficiency of a glycoprotein component of the dystrophin complex in dystrophic muscle
6. Mechanical function of dystrophin in muscle cells
7. Extent of shock-induced membrane leakage in human and mouse myotubes depends on dystrophin
8. Impaired metabolic modulation of α-adrenergic vasoconstriction in dystrophin-deficient skeletal muscle
9. Nitric oxide synthase complexed with dystrophin and absent from skeletal muscle sarcolemma in Duchenne muscular dystrophy
10. Neuronal nitric oxide synthase and dystrophin-deficient muscular dystrophy
11. Mdx muscle pathology is independent of nNOS perturbation
12. ATP-sensitive potassium channels mediate contraction-induced attenuation of sympathetic vasoconstriction in rat skeletal muscle
13. Mechanical loading regulates NOS expression and activity in developing and adult skeletal muscle
14. nNOS and eNOS modulate cGMP formation and vascular response in contracting fast-twitch skeletal muscle
15. Skeletal muscle contractions stimulate cGMP formation and attenuate vascular smooth muscle myosin phosphorylation via nitric oxide
16. Progressive muscular dystrophy in α-sarcoglycan deficient mice
17. Disruption of the β-sarcoglycan gene reveals pathogenetic complexity of limb-girdle muscular dystrophy type 2E
18. Disruption of the sarcoglycan-sacrospan complex in vascular smooth muscle: A novel mechanism for cardiomyopathy and muscular dystrophy