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Volumn 121, Issue 2, 2001, Pages 279-289

Hemodynamic unloading leads to regression of pulmonary vascular disease in rats

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL EXPERIMENT; ARTICLE; CONGENITAL HEART DISEASE; CONTROLLED STUDY; LUNG ARTERY PRESSURE; LUNG HEMODYNAMICS; NONHUMAN; PRIORITY JOURNAL; PULMONARY ARTERY; PULMONARY ARTERY DISEASE; PULMONARY HYPERTENSION; RAT;

EID: 0035130447     PISSN: 00225223     EISSN: None     Source Type: Journal    
DOI: 10.1067/mtc.2001.111657     Document Type: Article
Times cited : (58)

References (24)
  • 6
    • 0019205876 scopus 로고
    • Ultrastructural findings in lung biopsy material from children with congenital heart defects
    • (1980) Am J Pathol , vol.101 , pp. 527-542
    • Meyrick, B.1    Reid, L.2
  • 10
    • 0033612184 scopus 로고    scopus 로고
    • Regression of hypertrophied rat pulmonary arteries in organ culture is associated with suppression of proteolytic activity, inhibition of tenascin-C, and smooth muscle cell apoptosis
    • (1999) Circ Res , vol.84 , pp. 1223-1233
    • Cowan, K.N.1    Jones, P.L.2    Rabinovitch, M.3
  • 23
    • 0029806822 scopus 로고    scopus 로고
    • Tenascin-C induced with progressive pulmonary vascular disease in rats is functionally related to increased smooth muscle cell proliferation
    • (1996) Circ Res , vol.79 , pp. 1131-1142
    • Jones, P.L.1    Rabinovitch, M.2
  • 24
    • 0030860609 scopus 로고    scopus 로고
    • Regulation of tenascin-C, a vascular smooth muscle cell survival factor that interacts with the αvβ3 integrin to promote epidermal growth factor receptor phosphorylation and growth
    • (1997) J Cell Biol , vol.139 , pp. 279-293
    • Jones, P.L.1    Crack, J.2    Rabinovitch, M.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.