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Volumn 76, Issue 3, 2001, Pages 815-824
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Endogenously released DOPA is a causal factor for glutamate release and resultant delayed neuronal cell death by transient ischemia in rat striata
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Author keywords
Brain ischemia; Competitive DOPA antagonist; Delayed neuronal cell death; DOPA cyclohexyl ester; DOPA release; Glutamate release
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Indexed keywords
AROMATIC LEVO AMINO ACID DECARBOXYLASE;
DOPA;
DOPA ANTAGONIST;
DOPA CYCLOHEXYL ESTER;
DOPAMINE;
GLUTAMIC ACID;
NEUROPROTECTIVE AGENT;
NEUROTRANSMITTER;
UNCLASSIFIED DRUG;
AMINO ACID ANALYSIS;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
BRAIN ISCHEMIA;
CANNULATION;
CONTROLLED STUDY;
CORPUS STRIATUM;
DOPAMINE RELEASE;
MALE;
MICRODIALYSIS;
NERVE CELL NECROSIS;
NEUROPROTECTION;
NEUROTRANSMITTER RELEASE;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
STEREOTAXIC SURGERY;
ANIMALS;
CELL DEATH;
CORPUS STRIATUM;
DIHYDROXYPHENYLALANINE;
DOPAMINE;
ENZYME INHIBITORS;
GLUTAMIC ACID;
HYDRAZINES;
ISCHEMIC ATTACK, TRANSIENT;
LEVODOPA;
MALE;
NEURONS;
RATS;
RATS, WISTAR;
TIME FACTORS;
ANIMALIA;
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EID: 0035125530
PISSN: 00223042
EISSN: None
Source Type: Journal
DOI: 10.1046/j.1471-4159.2001.00068.x Document Type: Article |
Times cited : (26)
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References (49)
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