Role of cadherins and matrixins in melanoma

Current Opinion in Oncology

Volumn 13, Issue 2, 2001, Pages 117-123

  Gruss, Claus ^a   Herlyn, Meenhard ^a  

^a WISTAR INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CADHERIN; MATRIX METALLOPROTEINASE; MATRIX METALLOPROTEINASE INHIBITOR; UVOMORULIN;

CANCER GROWTH; CANCER SURVIVAL; CARCINOGENESIS; CELL TYPE; ENVIRONMENTAL FACTOR; HOMEOSTASIS; KERATINOCYTE; MALIGNANT TRANSFORMATION; MELANOMA; PRIORITY JOURNAL; REVIEW;

ANIMALS; CADHERINS; CELL ADHESION; GAP JUNCTIONS; HUMANS; KERATINOCYTES; MATRIX METALLOPROTEINASES; MELANOMA; NEOPLASM INVASIVENESS; SIGNAL TRANSDUCTION; SKIN NEOPLASMS; TISSUE INHIBITOR OF METALLOPROTEINASES;

EID: 0035076093     PISSN: 10408746     EISSN: None     Source Type: Journal    
DOI: 10.1097/00001622-200103000-00006     Document Type: Review

References (41)

1. T-cell recognition of melanoma-associated antigens
2. Heterogeneity in expression of human leukocyte antigens and melanoma-associated antigens in advanced melanoma
3. Cancer vaccination
4. Undifferentiated keratinocytes control growth, morphology, and antigen expression of normal melanocytes through cell-cell contact
5. E-cadherin expression in melanoma cells restores keratinocyte-mediated growth control and downregulates expression of invasion-related adhesion receptors
6. Cadherin repertoire determines partner-specific gap junctional communication during melanoma progression
7. E-cadherin downregulation in cancer: Fuel on the fire?
8. Human melanoma progression in skin reconstructs: Biological significance of bFGF
9. Shifts in cadherin profiles between human normal melanocytes and melanomas
10. E-cadherin is the major mediator of human melanocyte adhesion to keratinocytes in vitro
11. Alterations in cadherin and catenin expression during the biological progression of melanocytic tumours
12. Cadherin switching in human prostate cancer progression
13. Expression of N-cadherin by human squamous carcinoma cells induces a scattered fibroblastic phenotype with disrupted cell-cell adhesion
14. N-cadherin promotes adhesion between invasive breast cancer cells and the stroma
15. The role of the cell-adhesion molecule E-cadherin as a tumour-suppressor gene
16. Travel bulletin: Traffic jam cause cancer
17. Cooperative regulation of cell polarity and growth by drosophila tumor suppressors
18. Signal transduction of beta-catenin
19. Beta-catenin signaling and cancer
20. Beta-catenin regulates expression of cyclin D1 in colon carcinoma cells
21. Matrix metalloproteinases
22. Tissue inhibitors of metalloproteinases: Evolution, structure and function
23. Regulation of matrix metalloproteinase expression in tumor invasion
24. Transmembrane/cytoplasmic domain-mediated membrane-type-1 matrix metalloprotease docking to invadopodia is required for cell invasion
25. Regulation of membrane-type-1 matrix metalloproteinase activity by its cytoplasmic domain
26. Localization of matrix metalloproteinase MMP-2 to the surface of invasive cells by interaction with integrin alpha v beta 3
27. Disruption of angiogenesis by PEX, a noncatalytic metalloproteinase fragment with integrin binding activity
28. A novel host/tumor cell interaction activates matrix metalloproteinase 1 and mediates invasion through type I collagen
29. Control of type IV collagenase activity by components of the urokinase-plasmin system: A regulatory mechanism with cell-bound reactants
30. Metastatic behavior of human melanoma cell lines in nude mice correlates with urokinase-type plasminogen activator, its type-1 inhibitor, and urokinase-mediated matrix degradation
31. Tissue inhibitors of metalloproteinases: Structure, regulation and biological functions
32. Cell cycle-associated accumulation of tissue inhibitor of metalloproteinases-1 (TIMP-1) in the nuclei of human gingival fibroblasts
33. Tissue inhibitor of matrix metalloproteinase-2 regulates matrix metalloproteinase-2 activation by modulation of membrane-type-1 matrix metalloproteinase activity in high and low invasive melanoma cell lines
34. TIMP-3 induces cell death by stabilizing TNF-alpha receptors on the surface of human colon carcinoma cells
35. Adenovirus-mediated gene delivery of tissue inhibitor of metalloproteinases-3 inhibits invasion and induces apoptosis in melanoma cells
36. The activity of collagenase-1 is required for keratinocyte migration on a type I collagen matrix
37. Induction of cell migration by matrix metalloprotease-2 cleavage of laminin-5
38. Role of cell surface metalloprotease MT1-MMP in epithelial cell migration over laminin-5
39. Fibroblasts surrounding melanoma express elevated levels of matrix metalloproteinase-1 (MMP-1) and intercellular adhesion molecule-1 (ICAM-1) in vitro