A novel mechanism associated with idiopathic ventricular fibrillation (IVF) mutations R1232W and T1620M in human cardiac sodium channels

Pflugers Archiv European Journal of Physiology

Volumn 442, Issue 2, 2001, Pages 204-211

  Vilin, Yuriy Y ^a   Fujimoto, Esther ^a   Ruben, Peter C ^a  

^a UTAH STATE UNIVERSITY   (United States)

Author keywords

hNav1.5; Idiopathic ventricular fibrillation; Sodium channels; Xenopus oocytes

Indexed keywords

PROTEIN SUBUNIT; SODIUM CHANNEL;

ANIMAL CELL; ARTICLE; CELL MEMBRANE STEADY POTENTIAL; DISEASE ASSOCIATION; GENE MUTATION; HEART DEPOLARIZATION; HEART MUSCLE EXCITABILITY; HEART VENTRICLE ARRHYTHMIA; MEMBRANE CURRENT; NONHUMAN; PRIORITY JOURNAL; SODIUM CURRENT; STEADY STATE; XENOPUS LAEVIS;

ANIMALS; ELECTROPHYSIOLOGY; HOMEOSTASIS; HUMANS; KINETICS; MUTATION; MYOCARDIUM; OOCYTES; SODIUM CHANNELS; TIME FACTORS; VENTRICULAR FIBRILLATION; XENOPUS LAEVIS;

EID: 0035023665     PISSN: 00316768     EISSN: None     Source Type: Journal    
DOI: 10.1007/s004240100529     Document Type: Article

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