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Volumn 45, Issue 5, 2001, Pages 403-411

Effector mechanism and clinical response of BAK (BRM-activated killer) immuno-cell therapy for maintaining satisfactory QOL of advanced cancer patients utilizing CD56-positive NIE (Neuro-Immune-Endocrine) cells

Author keywords

CD56 positive cells; Immuno cell therapy; Prolonged NC; QOL

Indexed keywords

ALPHA INTERFERON; BETA ENDORPHIN; BIOLOGICAL RESPONSE MODIFIER; CD56 ANTIGEN; IMMUNOGLOBULIN; INTERLEUKIN 2;

EID: 0034993191     PISSN: 03855600     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1348-0421.2001.tb02638.x     Document Type: Article
Times cited : (10)

References (24)
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    • Neuromodulation of natural killer cell activity
    • Goetzl, E.J., and Spector, N.H. (eds), Neuroimmune networks, Alan R. Liss Inc., New York
    • (1989) , pp. 39-49
    • Arora, P.K.1
  • 3
    • 0024532757 scopus 로고
    • A molecular basis for bidirectional communication between the immune and neuroendocrine systems
    • (1989) Physiol. Rev. , vol.69 , pp. 1-32
    • Blalock, J.E.1
  • 6
    • 0028158838 scopus 로고
    • Antitumor effect of intratumoral administration of biological response modifiers: Induction of immunosuppressive acidic protein, a type of α1-acid glycoprotein, in mice
    • (1994) Jpn. J. Cancer Res. , vol.85 , pp. 93-100
    • Ebina, T.1    Murata, K.2    Tamura, K.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.