Interferon-γ production in skin during contact hypersensitivity. No contribution from keratinocytes [6]

Journal of Investigative Dermatology

Volumn 117, Issue 1, 2001, Pages 163-

  Akiba, H ^a   Ducluzeau, M T ^a   Nicolas, J F ^a  

^a INSERM   (France)

Author keywords

[No Author keywords available]

Indexed keywords

1 FLUORO 2,4 DINITROBENZENE; CD8 ANTIGEN; GAMMA INTERFERON; MESSENGER RNA; MONOCLONAL ANTIBODY;

ANIMAL TISSUE; CONTACT ALLERGY; CYTOKINE PRODUCTION; EPIDERMIS CELL; IMMUNOHISTOCHEMISTRY; KERATINOCYTE; LETTER; MOUSE; NONHUMAN; PRIORITY JOURNAL; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SKIN; SKIN INFLAMMATION; T LYMPHOCYTE;

EID: 0034943886     PISSN: 0022202X     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.0022-202X.2001.01384.x     Document Type: Letter

References (5)

1. Early molecular events in the induction phase of contact sensitivity
2. Epidermal keratinocyte production of interferon-gamma immunoreactive protein and mRNA is an early event in allergic contact dermatitis
3. Cytotoxicity is mandatory for CD8 (+) T cell-mediated contact hypersensitivity
4. Contact dermatitis I. Pathophysiology of contact sensitivity
5. Cell populations primed by hapten sensitization in contact sensitivity are distinguished by polarized patterns of cytokine production: Interferon gamma-producing (Tc1) effector CD8+ T cells and interleukin (II) 4/IL-10-producing (Th2) negative regulatory CD4+ T cells