Disturbed neural circuits in a subtype of chronic catatonic schizophrenia demonstrated by F-18-FDG-PET and F-18-DOPA-PET

Journal of Neural Transmission

Volumn 108, Issue 6, 2001, Pages 661-670

  Lauer, M ^a   Schirrmeister, H ^b   Gerhard, A ^b   Ellitok, E ^b   Beckmann, H ^a   Reske, S N ^b   Stober G ^a  

^a UNIVERSITY OF WÜRZBURG   (Germany)

^b UNIVERSITY OF ULM   (Germany)

Author keywords

Entorhinal; Leonhard classification; Limbic maldevelopment; Speech sluggish catatonia; Systematic catatonia

Indexed keywords

FLUORODEOXYGLUCOSE F 18;

ADULT; ARTICLE; CASE REPORT; CATATONIA; CONTROLLED STUDY; DOPAMINERGIC SYSTEM; FRONTAL CORTEX; HALLUCINATION; HUMAN; MENTAL DISEASE; NERVE CELL DIFFERENTIATION; PHENOTYPE; POSITRON EMISSION TOMOGRAPHY; PRIORITY JOURNAL; PSYCHOMOTOR DISORDER; PUTAMEN; SCHIZOPHRENIA;

ADULT; BRAIN; CHRONIC DISEASE; DIHYDROXYPHENYLALANINE; FLUORINE RADIOISOTOPES; FLUORODEOXYGLUCOSE F18; GLUCOSE; HUMANS; NEURAL PATHWAYS; RADIOPHARMACEUTICALS; SCHIZOPHRENIA, CATATONIC; SPEECH; TOMOGRAPHY, EMISSION-COMPUTED;

EID: 0034927820     PISSN: 03009564     EISSN: None     Source Type: Journal    
DOI: 10.1007/s007020170043     Document Type: Article

References (37)

1. Parallel organization of functionally segregated circuits linking basal ganglia and cortex
2. Hypofrontality in schizophrenia: Distributed dysfunctional circuits in neuroleptic-naive patients
3. Prenatal disturbances of nerve cell migration in the entorhinal region: A common vulnerability factor in functional psychoses?
4. Genetic heterogeneity in catatonic schizophrenia: A family study
5. The frontal lobes, basal ganglia and temporal lobes as sites for schizophrenia
6. Positron emission tomography studies of abnormal glucose metabolism in schizophrenia
7. Neurocircuitries and neurotransmitter interaction in schizophrenia
8. Limbic-cortical neuronal damage and the pathophysiology of schizophrenia
9. A review of periodic catatonia
10. Prenatal removal of frontal association cortex in the fetal rhesus monkey: Anatomical and functional consequences in postnatal life
11. Hypofrontality in schizophrenia: RIP
12. Neuropathology of schizophrenia: Cortex, thalamus, basal ganglia, and neurotransmitter-specific projection systems
13. Striatal dopamine receptors and transporters in monkeys with neonatal temporal limbic damage
14. Regional kinetic constants and cerebral metabolic rate for glucose in normal human volunteers determined by dynamic positron emission tomography of [18F]-2-fluoro-2-deoxy-D-glucose
15. Abnormalitites of cerebral blood flow distribution in patients with chronic schizophrenia
16. Clinical-neuropathological studies of developmental disorders in the limbic system in chronic schizophrenia
17. Prenatal developmental disturbances in the limbic allocortex in schizophrenics
18. Circumscript malformation and nerve cell alterations in the entorhinal cortex of schizophrenics
19. A neurohistological correlate of schizophrenia
20. The human striatum in schizophrenia. I. Increase in overall striatal volume density in schizophrenics
21. Decreased apoptosis in the pathogenesis of schizophrenia
22. Increased volume of the nucleus accumbens in schizophrenia
23. Patterns of cerebral blood flow in schizophrenia
24. Immediate effects of risperidone on cortico-striato-thalamic loops and the hippocampus
25. Increase in size of the caudate nucleus of the cat after a prenatal neocortical lesion
26. Plasma homovanillic acid concentrations in catatonia
27. Cell death during development of the nervous system
28. CA3 neuronal degeneration follows chronic entorhinal cortex lesions
29. Disorientation of the hippocampal pyramidal cell and its processes in the schizophrenic patient
30. Neuronale strukturanormalität im orbitofrontalen cortex bei schizophrenen
31. The functional neuroanatomy of hallucinations in schizophrenia
32. Periodic catatonia: A schizophrenic subtype with major gene effect and anticipation
33. Splitting schizophrenia: Periodic catatonia-susceptibility locus on chromosome 15q15
34. Cross-species studies of sensorimotor gating of the startle reflex
35. Implications of normal brain development for the pathogenesis of schizophrenia