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Volumn 21, Issue 8, 2001, Pages 937-944
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Interactions between the endothelium-derived relaxing factor/nitric oxide system and the endogenous opiate system in the modulation of cerebral and spinal vascular CO2 responsiveness
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Author keywords
Cerebral blood flow; CO2 responsiveness; Nitric oxide; Opioid peptides
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Indexed keywords
ARGININE;
BETA FUNALTREXAMINE;
CARBON DIOXIDE;
DELTA OPIATE RECEPTOR;
ENDORPHIN;
ENDOTHELIUM DERIVED RELAXING FACTOR;
MICROSPHERE;
MU OPIATE RECEPTOR;
N(G) NITROARGININE METHYL ESTER;
NALOXONE;
NALTRINDOLE;
NITRIC OXIDE;
NITRIC OXIDE SYNTHASE INHIBITOR;
OPIATE;
OPIATE ANTAGONIST;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
BLOOD VESSEL REACTIVITY;
BRAIN BLOOD FLOW;
BRAIN BLOOD VESSEL;
CAT;
CONTROLLED STUDY;
DRUG POTENTIATION;
HYPERCAPNIA;
HYPOCAPNIA;
ISOTOPE LABELING;
MALE;
MOLECULAR INTERACTION;
NONHUMAN;
PRIORITY JOURNAL;
SPINAL CORD;
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EID: 0034898646
PISSN: 0271678X
EISSN: None
Source Type: Journal
DOI: 10.1097/00004647-200108000-00006 Document Type: Article |
Times cited : (17)
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References (37)
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