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Volumn 2, Issue 8, 2001, Pages 739-745
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Contribution of the innate immune system to autoimmune myocarditis: A role for complement
a a,b a a a a a,b c a,b |
Author keywords
[No Author keywords available]
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Indexed keywords
BIOLOGICAL MARKER;
COMPLEMENT COMPONENT C3B RECEPTOR;
COMPLEMENT RECEPTOR;
COMPLEMENT RECEPTOR TYPE 2;
CYTOKINE;
HERMES ANTIGEN;
PADGEM PROTEIN;
UNCLASSIFIED DRUG;
ADULTHOOD;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
AUTOIMMUNE DISEASE;
AUTOIMMUNITY;
B LYMPHOCYTE ACTIVATION;
COMPLEMENT ACTIVATION;
CONTROLLED STUDY;
CYTOKINE PRODUCTION;
EXPERIMENTAL MODEL;
FEMALE;
HEART DILATATION;
HEART DISEASE;
HEART FAILURE;
IMMUNE RESPONSE;
IMMUNE SYSTEM;
INCIDENCE;
LYMPHOCYTE PROLIFERATION;
MALE;
MOUSE;
MYOCARDITIS;
NONHUMAN;
PATHOGENESIS;
PRIORITY JOURNAL;
PROTEIN ANALYSIS;
PROTEIN EXPRESSION;
REGULATORY MECHANISM;
T LYMPHOCYTE ACTIVATION;
T LYMPHOCYTE SUBPOPULATION;
ADULT;
ANIMALS;
AUTOIMMUNITY;
COMPLEMENT ACTIVATION;
COMPLEMENT SYSTEM PROTEINS;
FEMALE;
HUMANS;
LYMPHOCYTE ACTIVATION;
MICE;
MYOCARDITIS;
RECEPTORS, COMPLEMENT;
T-LYMPHOCYTES;
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EID: 0034891226
PISSN: 15292908
EISSN: None
Source Type: Journal
DOI: 10.1038/90686 Document Type: Article |
Times cited : (153)
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References (50)
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