Preliminary study of insulin resistance induced by neonatal monosodium glutamate treatment in normal Wistar rats

Chinese Pharmacological Bulletin

Volumn 17, Issue 2, 2001, Pages 181-185

  Ding, S Y ^a   Shen, Z F ^a   Xie, M Z ^a  

^a INSTITUTE OF MATERIA MEDICA   (China)

Author keywords

Insulin resistance; Insulin sensitizer; MSG rats; Obesity

Indexed keywords

CHOLESTEROL; FATTY ACID; GLUCOSE; GLUTAMATE SODIUM; INSULIN; PIOGLITAZONE; TRIACYLGLYCEROL;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CHOLESTEROL BLOOD LEVEL; CONTROLLED STUDY; DRUG EFFECT; DRUG EFFICACY; EXPERIMENTAL MODEL; FATTY ACID BLOOD LEVEL; FEMALE; GLUCONEOGENESIS; GLUCOSE BLOOD LEVEL; GLUCOSE METABOLISM; GLUCOSE TOLERANCE; INSULIN BLOOD LEVEL; INSULIN RESISTANCE; INSULIN SENSITIVITY; INSULIN TOLERANCE TEST; LIPID METABOLISM; MALE; NEWBORN; NONHUMAN; OBESITY; RAT; TRIACYLGLYCEROL BLOOD LEVEL;

EID: 0034830648     PISSN: 10011978     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (9)

1. Monosodium glutamate (MSG)-obese rats develop glucose intolerance and insulin resistance to peripheral glucose uptake
2. Neonatal monosodium glutamate treatment increases epididymal adipose tissue sensitivity to insulin in three-month old rats
3. Plasma lipoprotein of monosodiurn glutamate-induced obese rats
4. Effects of pioglitazone on glucose and lipid, metabolism in normal and insulin resistant animals
5. Effects of pioglitazone on glucose and lipid metabolism in Wistar fatty rats
6. Role of fatty acids in the pathogenesis of insulin resistance and NIDDM
7. Adipose expression of tumor necrosis factor-α: Direct role in obesity-linked insulin resistance
8. The expression of tumor necrosis factor in human adipose tissue. Regulation by obesity, weight loss, and relationship to lipoprotein lipase
9. Pretranslational suppression of a glucose transporter protein causes insulin resistance in adipocytes from patients with non-insulin-dependent diabetes mellitus and obesity