p16Ink4a and p19Arf: Terrible twins

Trends in Molecular Medicine

Volumn 7, Issue 11, 2001, Pages 489-

  Weitzman, Jonathan B ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

7,12 DIMETHYLBENZ[A]ANTHRACENE; CARCINOGEN; CYCLIN DEPENDENT KINASE; CYCLIN DEPENDENT KINASE 4; CYCLIN DEPENDENT KINASE 6; PROTEIN P16; PROTEIN P19; PROTEIN P53; RAS PROTEIN; RETINOBLASTOMA PROTEIN; UNCLASSIFIED DRUG;

ANIMAL CELL; CANCER INHIBITION; CANCER PREVENTION; CANCER SUSCEPTIBILITY; CELL CYCLE; CELL PROLIFERATION; ENZYME INHIBITION; FIBROBLAST; GENE LOCUS; LUNG ADENOMA; LYMPHOMA; MALIGNANT TRANSFORMATION; MELANOMA; MOUSE; NONHUMAN; NOTE; PROTEIN STABILITY; PROTEIN STRUCTURE; SENESCENCE; SKIN PAPILLOMA; SOFT TISSUE SARCOMA; THYMOCYTE; TUMOR SUPPRESSOR GENE;

EID: 0034764683     PISSN: 14714914     EISSN: None     Source Type: Journal    
DOI: 10.1016/s1471-4914(01)02190-6     Document Type: Note

References (3)

1. Ink4a confers susceptibility to metastatic melanoma in mice
2. Ink4a with retention of p19Arf predispose mice to tumorigenesis
3. ARF