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Volumn 478, Issue 1-2, 2000, Pages 109-112
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Crosstalk between estrogen receptor α and the aryl hydrocarbon receptor in breast cancer cells involves unidirectional activation of proteasomes
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Author keywords
Aryl hydrocarbon receptor; Breast cancer cell; Crosstalk; Degradation; Estrogen receptor ; Proteasome
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Indexed keywords
2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN;
ANTIESTROGEN;
AROMATIC HYDROCARBON RECEPTOR;
BENZYLOXYCARBONYLLEUCYLLEUCYLLEUCINAL;
CALPASTATIN;
CALPEPTIN;
CHLOROQUINE;
ESTRADIOL;
ESTROGEN RECEPTOR ALPHA;
PROTEASOME;
PROTEINASE INHIBITOR;
RECEPTOR PROTEIN;
ARTICLE;
BREAST CANCER;
CANCER CELL CULTURE;
CELL STRAIN MCF 7;
CONTROLLED STUDY;
HUMAN;
HUMAN CELL;
PRIORITY JOURNAL;
PROTEIN DEGRADATION;
SIGNAL TRANSDUCTION;
BREAST NEOPLASMS;
CYSTEINE ENDOPEPTIDASES;
DOWN-REGULATION;
ENVIRONMENTAL POLLUTANTS;
ENZYME ACTIVATION;
ESTRADIOL;
ESTROGEN RECEPTOR ALPHA;
ESTROGEN RECEPTOR MODULATORS;
HUMANS;
MULTIENZYME COMPLEXES;
PROTEASOME ENDOPEPTIDASE COMPLEX;
RECEPTOR CROSS-TALK;
RECEPTORS, ARYL HYDROCARBON;
RECEPTORS, ESTROGEN;
TETRACHLORODIBENZODIOXIN;
TUMOR CELLS, CULTURED;
MINK CELL FOCUS-FORMING VIRUS;
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EID: 0034726057
PISSN: 00145793
EISSN: None
Source Type: Journal
DOI: 10.1016/S0014-5793(00)01830-5 Document Type: Article |
Times cited : (98)
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References (34)
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