Actin cytoskeletal function is spared, but apoptosis is increased, in WAS patient hematopoietic cells

Blood

Volumn 95, Issue 4, 2000, Pages 1283-1292

  Rengan, Ramesh ^b   Ochs, Hans D ^b   Sweet, Leonard I ^b   Keil, Michael L ^b   Gunning, William T ^b   Lachant, Neil A ^b   Boxer, Laurence A ^b   Omann, Geneva M ^a  

^a VETERANS AFFAIRS MEDICAL CENTER   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE; CYTOCHALASIN B; CYTOSKELETON PROTEIN; F ACTIN; FAS ANTIGEN; UNCLASSIFIED DRUG; WISKOTT ALDRICH SYNDROME PROTEIN;

ACTIN POLYMERIZATION; ANTIGEN EXPRESSION; APOPTOSIS; ARTICLE; CLINICAL ARTICLE; CONTROLLED STUDY; CYTOARCHITECTURE; CYTOSKELETON; ENZYME ACTIVITY; GENE MUTATION; HEMATOPOIETIC CELL; HUMAN; HUMAN CELL; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; WISKOTT ALDRICH SYNDROME;

EID: 0034651932     PISSN: 00064971     EISSN: None     Source Type: Journal    
DOI: 10.1182/blood.v95.4.1283.004k44_1283_1292     Document Type: Article

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