Human frataxin maintains mitochondrial iron homeostasis in Saccharomyces cerevisiae

Human Molecular Genetics

Volumn 9, Issue 17, 2000, Pages 2523-2530

  Cavadini, Patrizia ^a,b   Gellera, Cinzia ^b   Patel, Pragna I ^c   Isaya, Grazia ^a  

^a MAYO CLINIC   (United States)

^b DEPARTMENT OF NEUROSURGERY   (Italy)

^c BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CELL PROTEIN; COMPLEMENTARY DNA; FRATAXIN; FREE RADICAL; IRON; IRON SULFUR PROTEIN; MITOCHONDRIAL DNA;

AMINO ACID SEQUENCE; ARTICLE; AUTOSOMAL RECESSIVE DISORDER; CELL PROTECTION; CLINICAL FEATURE; CYTOTOXICITY; DEGENERATIVE DISEASE; EUKARYOTE; FRIEDREICH ATAXIA; GENE MUTATION; GENETIC LINKAGE; HEART; HEART DISEASE; HOMEOSTASIS; HUMAN; HUMAN CELL; IRON METABOLISM; MITOCHONDRIAL RESPIRATION; NONHUMAN; OXIDATIVE STRESS; PATHOGENESIS; PHENOTYPE; POINT MUTATION; PRIORITY JOURNAL; PROTEIN DETERMINATION; PROTEIN PROCESSING; PROTEIN STABILITY; SACCHAROMYCES CEREVISIAE; SEQUENCE HOMOLOGY; SPECIES DIFFERENCE;

ATAXIA; EUKARYOTA; SACCHAROMYCES CEREVISIAE;

EID: 0034641851     PISSN: 09646906     EISSN: None     Source Type: Journal    
DOI: 10.1093/hmg/9.17.2523     Document Type: Article

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