|
|
Volumn 275, Issue 45, 2000, Pages 35291-35296
|
|
Functional analyses of three Csx/Nkx-2.5 mutations that cause human congenital heart disease
a
a
a
a
a
a
a
a
a
a
|
|
Author keywords
[No Author keywords available]
|
|
Indexed keywords
HOMEODOMAIN PROTEIN;
TRANSCRIPTION FACTOR;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
CELLULAR DISTRIBUTION;
CONGENITAL HEART DISEASE;
CONTROLLED STUDY;
GENE MUTATION;
HEART MUSCLE CELL;
HUMAN;
NONHUMAN;
PRIORITY JOURNAL;
PROMOTER REGION;
PROTEIN DNA BINDING;
RAT;
ANIMALS;
ANIMALS, NEWBORN;
APOPTOSIS;
ATRIAL NATRIURETIC FACTOR;
CELL NUCLEUS;
CELLS, CULTURED;
COS CELLS;
CYTOPLASM;
DNA-BINDING PROTEINS;
ELECTROPHORESIS, POLYACRYLAMIDE GEL;
GATA4 TRANSCRIPTION FACTOR;
GENE EXPRESSION REGULATION;
GENES, REPORTER;
GLUTATHIONE TRANSFERASE;
HEART DISEASES;
HEART SEPTAL DEFECTS, ATRIAL;
HOMEODOMAIN PROTEINS;
HUMANS;
IMMUNOHISTOCHEMISTRY;
IN SITU NICK-END LABELING;
MICROSCOPY, FLUORESCENCE;
MUTATION;
MYOCARDIUM;
NUCLEAR PROTEINS;
PHENOTYPE;
PLASMIDS;
PRECIPITIN TESTS;
PROMOTER REGIONS (GENETICS);
PROTEIN BINDING;
RATS;
RECEPTORS, PURINERGIC P1;
SERUM RESPONSE FACTOR;
TRANS-ACTIVATION (GENETICS);
TRANSCRIPTION FACTORS;
TRANSCRIPTION, GENETIC;
TRANSFECTION;
XENOPUS PROTEINS;
ANIMALIA;
MAMMALIA;
|
|
EID: 0034634624
PISSN: 00219258
EISSN: None
Source Type: Journal
DOI: 10.1074/jbc.M000525200 Document Type: Article |
|
Times cited : (57)
|
|
References (26)
|
