Transition of apoptotic resistant vascular smooth muscle cells to troptotic sensitive state is correlated with downregulation of c-FLIP

Journal of Vascular Research

Volumn 37, Issue 6, 2000, Pages 523-531

  Imanishi, Toshio ^a   Hano, Takuzo ^a   Nishio, Ichiro ^a   Liles, W Conrad ^b   Schwartz, Stephen M ^b   Han, David K M ^b  

^a WAKAYAMA MEDICAL UNIVERSITY   (Japan)

^b UNIVERSITY OF WASHINGTON   (United States)

Author keywords

Apoptosis; c FLIP; Fas signaling; Protein inhibitor; Vascular smooth muscle cell

Indexed keywords

CASPASE 3; CASPASE 8; FAS ANTIGEN; INTERLEUKIN 1BETA CONVERTING ENZYME; PROTEIN BCL 2; PROTEIN INHIBITOR;

APOPTOSIS; ARTICLE; CELL DEATH; CELL PROTECTION; DOSE TIME EFFECT RELATION; DOWN REGULATION; ENZYME ACTIVITY; HUMAN; HUMAN CELL; HUMAN TISSUE; INFLAMMATION; PRIORITY JOURNAL; PROTEIN EXPRESSION; SENSITIZATION; SIGNAL TRANSDUCTION; VASCULAR SMOOTH MUSCLE;

ANTIGENS, CD95; APOPTOSIS; ARABIDOPSIS PROTEINS; ARTERIOSCLEROSIS; BCL-X PROTEIN; CARRIER PROTEINS; CASP8 AND FADD-LIKE APOPTOSIS REGULATING PROTEIN; CASPASE 1; CASPASE 3; CASPASE 8; CASPASE 9; CASPASES; CELLS, CULTURED; CYCLOHEXIMIDE; ENZYME INDUCTION; ENZYME-LINKED IMMUNOSORBENT ASSAY; FAS LIGAND PROTEIN; FATTY ACID DESATURASES; GENE EXPRESSION REGULATION; GENES, BCL-2; HUMANS; IN SITU NICK-END LABELING; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MEMBRANE GLYCOPROTEINS; MUSCLE, SMOOTH, VASCULAR; PROTEIN SYNTHESIS INHIBITORS; PROTO-ONCOGENE PROTEINS C-BCL-2; RECOMBINANT FUSION PROTEINS; SIGNAL TRANSDUCTION;

EID: 0034515526     PISSN: 10181172     EISSN: None     Source Type: Journal    
DOI: 10.1159/000054085     Document Type: Article

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