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Volumn 157, Issue 3, 2000, Pages 905-918

Spontaneous classical pathway activation and deficiency of membrane regulators render human neurons susceptible to complement lysis

Author keywords

[No Author keywords available]

Indexed keywords

CD59 ANTIGEN; COMPLEMENT; COMPLEMENT COMPONENT C1Q; COMPLEMENT COMPONENT C3B; COMPLEMENT COMPONENT C3B RECEPTOR; COMPLEMENT COMPONENT C4; COMPLEMENT COMPONENT C9; COMPLEMENT MEMBRANE ATTACK COMPLEX; DECAY ACCELERATING FACTOR; MEMBRANE COFACTOR PROTEIN; MEMBRANE PROTEIN;

EID: 0034495294     PISSN: 00029440     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0002-9440(10)64604-4     Document Type: Article
Times cited : (132)

References (65)
  • 31
    • 0024370744 scopus 로고
    • Oligodendrocytes and oligodendrocyte/type-2 astrocyte progenitor cells of adult rats are specifically susceptible to the lytic effects of complement in the absence of antibody
    • (1989) Proc Natl Acad Sci USA , vol.86 , pp. 9025-9029
    • Wren, D.R.1    Noble, M.2
  • 32
    • 0027139116 scopus 로고
    • Killing of the rat glial cells by complement: Deficiency of the rat analogue of CD59 is the cause of oligodendrocyte susceptibility to lysis
    • (1993) J Neuroimmunol , vol.48 , pp. 169-176
    • Piddlesden, S.J.1    Morgan, B.P.2
  • 50
    • 0023226359 scopus 로고
    • Primary cultures of murine astrocyte produce C3 and factor B, two components of the alternative pathway of complement activation
    • (1987) J Immunol , vol.139 , pp. 2361-2366
    • Levi-Strauss, M.1    Mallat, M.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.