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Volumn 20, Issue 6 B, 2000, Pages 4463-4471
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Restoration of wild-type p53 activity enhances the sensitivity of pleural metastasis to cisplatin through an apoptotic mechanism
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Author keywords
galactosidase; Cationic liposomes; Cisplatin; Lung cancer cell lines; Primary pleural metastatic cells; Tumor suppressor gene
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Indexed keywords
ANTINEOPLASTIC AGENT;
CISPLATIN;
PROTEIN P53;
PROTEIN P53 SN3;
UNCLASSIFIED DRUG;
VIRUS VECTOR;
APOPTOSIS;
ARTICLE;
CLINICAL ARTICLE;
DRUG EFFICACY;
GENE THERAPY;
GENE TRANSFER;
HUMAN;
HUMAN CELL;
HUMAN TISSUE;
PLEURA TUMOR;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
TUMOR SUPPRESSOR GENE;
ANTINEOPLASTIC AGENTS;
BETA-GALACTOSIDASE;
CARCINOMA, BRONCHOGENIC;
CARCINOMA, NON-SMALL-CELL LUNG;
CARCINOMA, SQUAMOUS CELL;
CISPLATIN;
DRUG RESISTANCE, NEOPLASM;
FEASIBILITY STUDIES;
GENES, P53;
LAC OPERON;
LIPOSOMES;
LUCIFERASES;
LUNG NEOPLASMS;
NEOPLASM PROTEINS;
PLEURAL EFFUSION;
TRANSFECTION;
TUMOR CELLS, CULTURED;
TUMOR SUPPRESSOR PROTEIN P53;
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EID: 0034484516
PISSN: 02507005
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
Times cited : (12)
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References (37)
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