How calcium influx through calcium leak channels is responsible for the elevated levels of calcium-dependent proteolysis in dystrophic myotubes

Trends in Cardiovascular Medicine

Volumn 10, Issue 6, 2000, Pages 268-272

  Alderton, J M ^a   Steinhardt, R A ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM; CALCIUM CHANNEL; CALPAIN; DEFLAZACORT; DYSTROPHIN; PREDNISOLONE;

ANIMAL MODEL; CALCIUM CELL LEVEL; CALCIUM TRANSPORT; CELL DAMAGE; CELL DEATH; CYTOSKELETON; DUCHENNE MUSCULAR DYSTROPHY; EXTRACELLULAR MATRIX; HOMEOSTASIS; HUMAN; MECHANICAL STRESS; MOUSE; MUSCLE CELL; MUSCULAR DYSTROPHY; MYOTUBE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; REVIEW; SARCOLEMMA;

ANIMALS; CALCIUM; CALCIUM CHANNELS; CALPAIN; CELLS, CULTURED; CYTOPLASM; DYSTROPHIN; HOMEOSTASIS; HUMANS; HYDROLYSIS; MUSCLE FIBERS; MUSCLE PROTEINS; MUSCULAR DYSTROPHY, DUCHENNE; SARCOLEMMA;

EID: 0034468908     PISSN: 10501738     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1050-1738(00)00075-X     Document Type: Review

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