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Volumn 29, Issue 9, 2000, Pages 801-813
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Mitochondrial or cytosolic catalase reverses the MnSOD-dependent inhibition of proliferation by enhancing respiratory chain activity, net ATP production, and decreasing the steady state levels of H2O2
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Author keywords
Catalase; Free radicals; Hydrogen peroxide; Manganese superoxide dismutase; Mitochondria
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Indexed keywords
ADENOSINE TRIPHOSPHATE;
CATALASE;
HYDROGEN PEROXIDE;
MANGANESE SUPEROXIDE DISMUTASE;
ANIMAL CELL;
ARTICLE;
CANCER;
CELL CULTURE;
CELL PROLIFERATION;
CELL TRANSFORMATION;
CONTROLLED STUDY;
CYTOSOL;
ENZYME ACTIVITY;
FIBROSARCOMA;
FLOW CYTOMETRY;
GENE OVEREXPRESSION;
IMMUNOFLUORESCENCE;
IN VITRO STUDY;
METABOLIC REGULATION;
MITOCHONDRION;
NONHUMAN;
OXYGEN CONSUMPTION;
PHENOTYPE;
PRIORITY JOURNAL;
RESPIRATORY CHAIN;
TUMOR CELL LINE;
ADENOSINE TRIPHOSPHATE;
CATALASE;
CELL DIVISION;
CYTOSOL;
ELECTRON TRANSPORT;
FREE RADICALS;
HUMAN;
HYDROGEN PEROXIDE;
MICROSCOPY, FLUORESCENCE;
MITOCHONDRIA;
SUPEROXIDE DISMUTASE;
SUPPORT, U.S. GOV'T, P.H.S.;
TRANSFECTION;
TUMOR CELLS, CULTURED;
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EID: 0034333151
PISSN: 08915849
EISSN: None
Source Type: Journal
DOI: 10.1016/S0891-5849(00)00362-2 Document Type: Article |
Times cited : (115)
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References (51)
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