Activation of caspase-3 and c-Jun NH2-terminal kinase-1 signaling pathways in tamoxifen-induced apoptosis of human breast cancer cells

Cancer Research

Volumn 60, Issue 21, 2000, Pages 5995-6000

  Mandlekar, Sandhya ^a,c   Yu, Rong ^a   Tan, Tse Hua ^b   Kong, Ah Ng Tony ^a  

^a UNIVERSITY OF ILLINOIS AT CHICAGO   (United States)

^b BAYLOR COLLEGE OF MEDICINE   (United States)

^c DUPONT COMPANY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ACETYLCYSTEINE; ALPHA TOCOPHEROL; ANTIOXIDANT; CASPASE 3; ENZYME INHIBITOR; ESTROGEN RECEPTOR; GLUTATHIONE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; STRESS ACTIVATED PROTEIN KINASE; TAMOXIFEN; TETRAPEPTIDE;

APOPTOSIS; ARTICLE; BREAST CANCER; CANCER CELL CULTURE; CHROMATIN CONDENSATION; DRUG MECHANISM; ENZYME ACTIVATION; HUMAN; HUMAN CELL; MEMBRANE DAMAGE; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN DEGRADATION; SIGNAL TRANSDUCTION;

AMINO ACID CHLOROMETHYL KETONES; ANTINEOPLASTIC AGENTS, HORMONAL; APOPTOSIS; BREAST NEOPLASMS; CASPASE 3; CASPASES; CYSTEINE PROTEINASE INHIBITORS; DRUG INTERACTIONS; ENZYME ACTIVATION; ENZYME INDUCTION; HUMANS; MAP KINASE SIGNALING SYSTEM; MITOGEN-ACTIVATED PROTEIN KINASE 8; MITOGEN-ACTIVATED PROTEIN KINASES; OLIGOPEPTIDES; TAMOXIFEN; TUMOR CELLS, CULTURED;

EID: 0034326818     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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