Mice mutant for Egfr and Shp2 have defective cardiac semilunar valvulogenesis

Nature Genetics

Volumn 24, Issue 3, 2000, Pages 296-299

  Chen, Binbin ^a   Bronson, Roderick T ^b   Klaman, Lori D ^a   Hampton, Thomas G ^a   Wang, Ju Feng ^a   Green, Patricia J ^c   Magnuson, Terry ^c   Douglas, Pamela S ^a   Morgan, James P ^a   Neel, Benjamin G ^a  

^a HARVARD MEDICAL SCHOOL   (United States)

^b TUFTS UNIVERSITY   (United States)

^c CASE WESTERN RESERVE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

EPIDERMAL GROWTH FACTOR RECEPTOR; PROTEIN TYROSINE PHOSPHATASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTA STENOSIS; AORTA VALVE DISEASE; ARTICLE; CONTROLLED STUDY; GENE MUTATION; HEART DEVELOPMENT; HEART HEMODYNAMICS; HEART VALVE; HEART VALVE REGURGITATION; HETEROZYGOSITY; MOUSE; NEWBORN; NONHUMAN; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ABNORMALITIES, MULTIPLE; ANIMALS; AORTIC VALVE; AORTIC VALVE INSUFFICIENCY; AORTIC VALVE STENOSIS; EPISTASIS, GENETIC; GENOTYPE; HEART CONDUCTION SYSTEM; HYPERPLASIA; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MESODERM; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; PROTEIN-TYROSINE-PHOSPHATASE; PULMONARY VALVE; RECEPTOR, EPIDERMAL GROWTH FACTOR; SEQUENCE DELETION; SHP1 PROTEIN TYROSINE PHOSPHATASE; VENTRICULAR DYSFUNCTION, LEFT;

ANIMALIA; RODENTIA;

EID: 0034091647     PISSN: 10614036     EISSN: None     Source Type: Journal    
DOI: 10.1038/73528     Document Type: Article

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