Stress- and growth-related gene expression are independent of chemical- induced prostaglandin E2 synthesis in renal epithelial cells

Chemical Research in Toxicology

Volumn 13, Issue 2, 2000, Pages 111-117

  Towndrow, Kelly M ^a   Mertens, Jozef J W M ^a,b   Jeong, Jeongmi K ^a,c   Weber, Thomas J ^a,d   Monks, Terrence J ^a   Lau, Serrine S ^a  

^a UNIVERSITY OF TEXAS AT AUSTIN   (United States)

^b WIL RESEARCH LABORATORIES   (United States)

^c Korea National Institute of Health   (South Korea)

^d PACIFIC NORTHWEST NATIONAL LABORATORY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CATALASE; HYDROQUINONE; HYDROQUINONE DERIVATIVE; PROSTAGLANDIN E2; PROSTAGLANDIN SYNTHASE;

ARTICLE; CELL PROLIFERATION; CONTROLLED STUDY; FIBROBLAST; GENE EXPRESSION REGULATION; HELA CELL; HUMAN; HUMAN CELL; KIDNEY EPITHELIUM; NEPHROTOXICITY; OXIDATION REDUCTION REACTION; PROSTAGLANDIN SYNTHESIS; STRESS; TOXICOKINETICS;

ANIMALS; CCAAT-ENHANCER-BINDING PROTEINS; CELL SURVIVAL; CELLS, CULTURED; DINOPROSTONE; DNA-BINDING PROTEINS; EPITHELIAL CELLS; GENE EXPRESSION; GLUTATHIONE; HSP70 HEAT-SHOCK PROTEINS; HYDROQUINONES; KIDNEY TUBULES, PROXIMAL; RNA, MESSENGER; SWINE; TRANSCRIPTION FACTOR CHOP; TRANSCRIPTION FACTORS;

EID: 0034011712     PISSN: 0893228X     EISSN: None     Source Type: Journal    
DOI: 10.1021/tx990160s     Document Type: Article

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