Fus deficiency in mice results in defective B-lymphocyte development and activation, high levels of chromosomal instability and perinatal death

Nature Genetics

Volumn 24, Issue 2, 2000, Pages 175-179

  Hicks, Geoffrey G ^a   Singh, Nagendra ^b   Nashabi, Abudi ^b   Mai, Sabine ^a   Bozek, Gracjan ^a   Klewes, Ludger ^a   Arapovic, Djula ^a   White, Erica K ^b   Koury, Mark J ^b   Oltz, Eugene M ^b   Van Kaer, Luc ^b   Ruley, H Earl ^b  

^a UNIVERSITY OF MANITOBA   (Canada)

^b VANDERBILT UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

TRANSCRIPTION FACTOR;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; B LYMPHOCYTE ACTIVATION; CELL MATURATION; CELL PROLIFERATION; CELL VIABILITY; CHIMERA; DNA BINDING; GENE TRANSLOCATION; GENETIC STABILITY; IN VIVO STUDY; MOUSE; NONHUMAN; PERINATAL DEATH; PRIORITY JOURNAL; PROTEIN DOMAIN;

ANIMALS; ANIMALS, NEWBORN; B-LYMPHOCYTES; BONE MARROW CELLS; CHIMERA; CROSSES, GENETIC; FEMALE; GENOTYPE; HETEROGENEOUS-NUCLEAR RIBONUCLEOPROTEINS; HUMANS; LIVER; LYMPHOCYTE ACTIVATION; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; RESTRICTION MAPPING; RIBONUCLEOPROTEINS; RNA-BINDING PROTEIN FUS; RNA-BINDING PROTEINS; SPLEEN;

ANIMALIA;

EID: 0033968408     PISSN: 10614036     EISSN: None     Source Type: Journal    
DOI: 10.1038/72842     Document Type: Article

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