BCR-ABL prevents c-Jun-mediated and proteasome-dependent FUS (TLS) proteolysis through a protein kinase CβII-dependent pathway

Molecular and Cellular Biology

Volumn 20, Issue 16, 2000, Pages 6159-6169

  Perrotti, D ^a   Iervolino, A ^a   Cesi, V ^a   Cirinna, M ^a   Lombardini, S ^a   Grassilli, E ^a   Bonatti, S ^a   Claudio, P P ^a   Calabretia, B ^a  

^a THOMAS JEFFERSON UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BCR ABL PROTEIN; HETEROGENEOUS NUCLEAR RIBONUCLEOPROTEIN; PROTEASOME; PROTEIN KINASE C; STRESS ACTIVATED PROTEIN KINASE;

AMINO ACID SUBSTITUTION; ANIMAL CELL; ARTICLE; CONTROLLED STUDY; DNA BINDING; ENZYME PHOSPHORYLATION; GENE CONTROL; GENE EXPRESSION; GENE INDUCTION; GENE REPRESSION; GENE TRANSLOCATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN PROCESSING; PROTEIN PROTEIN INTERACTION; TRANSACTIVATION;

ANIMALS; APOPTOSIS; CELL LINE; CYSTEINE ENDOPEPTIDASES; ENZYME ACTIVATION; FUSION PROTEINS, BCR-ABL; GENES, JUN; HETEROGENEOUS-NUCLEAR RIBONUCLEOPROTEIN GROUP A-B; HETEROGENEOUS-NUCLEAR RIBONUCLEOPROTEINS; MICE; MULTIENZYME COMPLEXES; PHOSPHORYLATION; PROTEASOME ENDOPEPTIDASE COMPLEX; PROTEIN KINASE C; RIBONUCLEOPROTEINS; RNA-BINDING PROTEIN FUS; SIGNAL TRANSDUCTION;

ANIMALIA;

EID: 0033858675     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.20.16.6159-6169.2000     Document Type: Article

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