Many pathways to cardiac hypertrophy

Journal of Molecular and Cellular Cardiology

Volumn 32, Issue 8, 2000, Pages 1381-1384

  Steinberg, Susan F ^a  

^a New York Presbyterian Columbia University Medical Center   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCINEURIN; MITOGEN ACTIVATED PROTEIN KINASE; STRESS ACTIVATED PROTEIN KINASE;

EDITORIAL; HEART MUSCLE CONTRACTILITY; HEART PACING; HEART PERFORMANCE; HEART VENTRICLE HYPERTROPHY; HUMAN; PATHOGENESIS; PRIORITY JOURNAL;

EID: 0033852461     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1006/jmcc.2000.1202     Document Type: Editorial

References (26)

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4. Signaling properties and functions of two distinct cardiomyocyte protease-activated receptors
5. Induction of atrial natriuretic factor and myosin light chain-2 gene expression in cultured ventricular myocytes by electrical stimulation of contraction
6. Acceleration of growth of cultured cardiomyocytes and translocation of protein kinase C
7. Involvement of cytoplasmic calcium and protein kinases in the regulation of atrial natriuretic factor secretion by contraction rate and endothelin
8. Electrical stimulation of neonatal cardiac myocytes activates the NFAT3 and GATA4 pathways and up-regulates the adenylosuccinate synthetase 1 gene
9. Electrical stimulation of neonatal cardiomyocytes results in the sequential activation of nuclear genes governing mitochondrial proliferation and diferentiation
10. Activation of the cytochrome c gene by electrical stimulation in neonatal rat cardiac myocytes. Role of NRF-1 and c-Jun
11. Isoenzyme-specific protein kinase C and c-Jun N-terminal kinase activation by electrically stimulated contraction of neonatal rat ventricular myocytes
12. Expression of a constitutively activated mutant of the β-isozyme of protein kinase C in cardiac myocytes stimulates the promoter of the β-myosin heavy chain isogene
13. Expression of protein kinase C-β in the heart causes hypertrophy in adult mice and sudden death in neonates
14. Classical, novel and atypical isoforms of PKC stimulate ANF- and TRE/AP-1-regulated-promoter activity in ventricular cardiomyocytes
15. Targeted over expression of protein kinase C β2 isoform in myoardium causes cardiomyopathy
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17. 3 generation
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24. Activation of p38 and c-Jun N-terminal kinase pathways and induction of apoptosis by chelerythrine do not require inhibition of protein kinase C
25. Cardiac muscle cell hypertrophy and apoptosis induced by distinct members of the p38 mitogen-activated protein kinase family
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