[Arg6, D-Trp7,9, NmePhe8]-substance P (6-11) (antagonist G) induces AP-1 transcription and sensitizes cells to chemotherapy

British Journal of Cancer

Volumn 83, Issue 7, 2000, Pages 941-948

  MacKinnon, A C ^a   Waters, C ^b   Rahman, I ^a   Harani, N ^a   Rintoul, R ^a   Haslett, C ^a   Sethi, T ^a  

^a UNIVERSITY OF EDINBURGH   (United Kingdom)

^b WESTERN GENERAL HOSPITAL   (United Kingdom)

Author keywords

Antagonist G; AP 1; Apoptosis; Chemosensitivity; ROS; SCLC

Indexed keywords

ACETYLCYSTEINE; CERAMIDE; ETOPOSIDE; GLUTATHIONE; NEUROPEPTIDE RECEPTOR; REACTIVE OXYGEN METABOLITE; STRESS ACTIVATED PROTEIN KINASE; SUBSTANCE P ANTAGONIST; SUBSTANCE P [6-11]; TRANSCRIPTION FACTOR AP 1;

ANOXIA; APOPTOSIS; ARTICLE; CANCER CELL; CANCER GROWTH; CANCER INHIBITION; CHEMOSENSITIVITY; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME ACTIVITY; HUMAN; HUMAN CELL; LIPID PEROXIDATION; LUNG SMALL CELL CANCER; OXIDATION REDUCTION REACTION; PRIORITY JOURNAL;

EID: 0033834860     PISSN: 00070920     EISSN: None     Source Type: Journal    
DOI: 10.1054/bjoc.2000.1362     Document Type: Article

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