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Volumn 157, Issue 4, 2000, Pages 1393-1403

p57(KIP2) is not mutated in hepatoblastoma but shows increased transcriptional activity in a comparative analysis of the three imprinted genes p57(KIP2), IGF2, and H19

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIN DEPENDENT KINASE INHIBITOR; GENE PRODUCT; MESSENGER RNA;

EID: 0033815532     PISSN: 00029440     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0002-9440(10)64652-4     Document Type: Article
Times cited : (43)

References (63)
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    • Establishment of a cell line and its clonal sublines from a patient with hepatoblastoma
    • (1976) Gann , vol.67 , pp. 1-10
    • Doi, I.1
  • 36
    • 0028284563 scopus 로고
    • Genomic imprinting: Mechanism and role in human pathology
    • (1994) Am J Pathol , vol.144 , pp. 431-443
    • Tycko, B.1
  • 42
    • 0030670449 scopus 로고    scopus 로고
    • Competitiona - A common motif for the imprinting mechanism?
    • (1997) EMBO J , vol.16 , pp. 6899-6905
    • Barlow, D.P.1
  • 58
    • 0029033861 scopus 로고
    • The retinoblastoma protein and cell cycle control
    • (1995) Cell , vol.81 , pp. 323-330
    • Weinberg, R.A.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.