To be or Notch to be

Nature Medicine

Volumn 6, Issue 11, 2000, Pages 1210-1211

  Brenner, M ^a  

^a CENTER FOR CELL AND GENE THERAPY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CELL DIFFERENTIATION; CELL IMMORTALIZATION; DROSOPHILA; HEMATOPOIESIS; HEMATOPOIETIC STEM CELL; NONHUMAN; PRIORITY JOURNAL; SHORT SURVEY; SIGNAL TRANSDUCTION;

ANIMALS; CELL DIFFERENTIATION; HEMATOPOIETIC STEM CELLS; HUMANS; LEUKOPOIESIS; MEMBRANE PROTEINS; RECEPTOR, NOTCH1; RECEPTORS, CELL SURFACE; SIGNAL TRANSDUCTION; T-LYMPHOCYTES; TRANSCRIPTION FACTORS;

EID: 0033694293     PISSN: 10788956     EISSN: None     Source Type: Journal    
DOI: 10.1038/81297     Document Type: Short Survey

References (10)

1. Pluripotent cytokine dependent hematopoietic stem cells are immortalized by constitutive Notch1 signaling
2. Hox homeobox genes as regulators of normal and malignant hematopoiesis
3. Notch1 signaling
4. Making a difference: The role of cell-cell interactions in establishing separate identities for equivalent cells
5. Notch 1 in vertebrates
6. The LIN-12/Notch1 signaling pathway and its regulation
7. Stromal expression of Jagged 1 promotes colony formation by fetal hematopoietic progenitor cells
8. Deficient T cell fate specification in mice with an induced inactivation of Notch1
9. Exclusive development of T cell neoplasms in mice transplanted with bone marrow expressing activated Notch1 alleles
10. Regulation of T cell fate by Notch1