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Volumn 49, Issue 11, 2000, Pages 610-618
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Mechanism of phosphatidylinositol 3-kinase-dependent increases in BAC1.2F5 macrophage-like cell density in response to M-CSF: Phosphatidylinositol 3-kinase inhibitors increase the rate of apoptosis rather than inhibit DNA synthesis
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Author keywords
Apoptosis; C fms; M CSF; PI 3 kinase; Proliferation
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Indexed keywords
2 MORPHOLINO 8 PHENYLCHROMONE;
COLONY STIMULATING FACTOR 1;
DNA;
PHOSPHATIDYLINOSITOL 3 KINASE;
WORTMANNIN;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
CELL COUNT;
CELL DENSITY;
CELL PROLIFERATION;
CELL PROTECTION;
CELL STIMULATION;
CELL SURVIVAL;
DNA SYNTHESIS;
ENZYME ACTIVITY;
MACROPHAGE;
MOUSE;
NONHUMAN;
1-PHOSPHATIDYLINOSITOL 3-KINASE;
ANDROSTADIENES;
ANIMALS;
APOPTOSIS;
CELL COUNT;
CELL DIVISION;
CELL LINE;
CELL SURVIVAL;
CHROMONES;
CULTURE MEDIA, SERUM-FREE;
DNA REPLICATION;
ENZYME INHIBITORS;
MACROPHAGE COLONY-STIMULATING FACTOR;
MACROPHAGES;
MICE;
MORPHOLINES;
PROTEIN-SERINE-THREONINE KINASES;
PROTO-ONCOGENE PROTEINS;
PROTO-ONCOGENE PROTEINS C-AKT;
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EID: 0033679303
PISSN: 10233830
EISSN: None
Source Type: Journal
DOI: 10.1007/s000110050638 Document Type: Article |
Times cited : (15)
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References (36)
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