Regulation of TNFRSF6 (FAS) expression in ataxia telangiectasia cells by ionizing radiation

Radiation Research

Volumn 154, Issue 6, 2000, Pages 616-624

  Albanese, J ^a   Dainiak, N ^a  

^a BRIDGEPORT HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 3; CASPASE 8; CASPASE INHIBITOR; FAS ANTIGEN; LIPOCORTIN 5;

ARTICLE; ATAXIA TELANGIECTASIA; CELL DEATH; CONTROLLED STUDY; CYTOTOXICITY; ENZYME ACTIVATION; FLOW CYTOMETRY; GENE EXPRESSION REGULATION; HUMAN; HUMAN CELL; IONIZING RADIATION; LYMPHOCYTE; PRIORITY JOURNAL; RADIOSENSITIVITY;

ATAXIA; ATAXIA TELANGIECTASIA;

EID: 0033658372     PISSN: 00337587     EISSN: None     Source Type: Journal    
DOI: 10.1667/0033-7587(2000)154[0616:ROTFEI]2.0.CO;2     Document Type: Article

References (31)

1. Clustering of missense mutations in the ataxia-telangiectasia gene in a sporadic T-cell leukemia
2. Testing the role of p 53 in the expression of genetic instability and apoptosis in ataxia telangiectasia
3. Elevated frequency of p53-independent apoptosis after irradiation increases levels of DNA breaks in ataxia telangiectasia lymphoblasts
4. Defective control of apoptosis, radiosensitivity, and spindle checkpoint in ataxia telangiectasia
5. Ataxia telangiectasia and cellular responses to DNA damage
6. p53 and ATM: Cell cycle, cell death and cancer
7. Deregulated apoptosis in ataxia telangiectasia: Association with clinical stigmata and radiosensitivity
8. Apoptosis by death factor
9. FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis
10. A novel protein that interacts with the death domain of Fas/APO- 1 contains a sequence motif related to the death domain
11. Cytotoxicity-dependent APO-1 (Fas/CD95)-associated proteins form a death-inducing signaling complex (DISC) with the receptor
12. Caspases: Enemies within
13. Radiation-induced apoptosis in human lymphocytes and lymphoma cells critically relies on the up-regulation of CD95/Fas/APO- 1 ligand
14. Biologically active Fas antigen and its cognate ligand are expressed on plasma membrane-derived extracellular vesicles
15. Mutations of CD95 (Fas/Apo-1) gene in adult T-cell leukemia cells
16. Dysregulation of CD95/CD95 ligand-apoptotic pathway in CD3(+) large granular lymphocyte leukemia
17. Human ICE/CED-3 protease nomenclature
18. Radiation and stress-induced apoptosis: A role for Fas/Fas ligand interactions
19. Markedly diminished radiation-induced lymphocyte apoptosis in lpr mice suggests a role for Fas in eliminating damaged cells
20. Chromosome aberrations scoring for biological dosimetry in a criticality accident
21. The tyrosine kinase Lck is required for CD95-independent caspase- 8 activation and apoptosis in response to ionizing radiation
22. Differential role of caspase-8 and BID activation during radiation- and CD95-induced apoptosis
23. Activation of the CD95 (APO-1/Fas) pathway in drug- and gamma-irradiation-induced apoptosis of brain tumor cell
24. Radiation-induced expression of functional Fas ligand in EBV-positive human nasopharyngeal carcinoma cells
25. Up-regulation of Fas (CD95) in human p53(wild-type) cancer cells treated with ionizing radiation
26. Molecular mechanisms of ionizing radiation-induced apoptosis
27. Human DNA-activated protein kinase phosphorylates serine 15 and 37 in the amino-terminal transactivation domain of human p53
28. Silencer and enhancer regions in the human CD95 (Fas/APO-1) gene with sequence similarity to the granulocyte-macrophage colony-stimulating factor promoter: Binding of single strand-specific silencer factors and AP-1 and NF-AT-like enhancer factors
29. Damage-sensing mechanisms in human cells after ionizing radiation
30. Ordering of ceramide formation caspase activation and mitochondrial changes during CD95- and DNA damage-induced apoptosis
31. Impaired regulation of nuclear factor κB results in apoptosis induced by gamma radiation