Redox manipulation of NMDA receptors in vivo: Alteration of acute pain transmission and dynorphin-induced allodynia

Pain

Volumn 80, Issue 1-2, 1999, Pages 37-43

  Laughlin, T M ^a   Kitto, K F ^a   Wilcox, G L ^a  

^a UNIVERSITY OF MINNESOTA   (United States)

Author keywords

Dynorphin; Hyperalgesia; N methyl D aspartate receptor; Neuropathic pain; Redox; Spinal cord

Indexed keywords

5,5' DITHIOBIS(2 NITROBENZOIC ACID); ALPHA AMINO 3 HYDROXY 5 METHYL 4 ISOXAZOLEPROPIONIC ACID; DITHIOTHREITOL; DYNORPHIN; GLUTAMIC ACID; N METHYL DEXTRO ASPARTIC ACID RECEPTOR;

AGED; ALLODYNIA; ANIMAL EXPERIMENT; ARTICLE; BEHAVIOR; CONTROLLED STUDY; DRUG EFFECT; HUMAN TISSUE; HYPERALGESIA; INTRATHECAL DRUG ADMINISTRATION; MALE; MODULATION; MOUSE; NERVE CELL PLASTICITY; NERVE INJURY; NEURALGIA; NOCICEPTION; NONHUMAN; PAIN THRESHOLD; PRIORITY JOURNAL; SPINAL CORD;

ACUTE DISEASE; ANIMALS; DITHIONITROBENZOIC ACID; DITHIOTHREITOL; DYNORPHINS; EXCITATORY AMINO ACID AGONISTS; HYPERALGESIA; INJECTIONS, SPINAL; MALE; MICE; MICE, INBRED ICR; N-METHYLASPARTATE; OXIDATION-REDUCTION; PAIN; PAIN MEASUREMENT; RECEPTORS, N-METHYL-D-ASPARTATE; REDUCING AGENTS; SIGNAL TRANSDUCTION; SPINAL CORD; SULFHYDRYL REAGENTS; SYNAPTIC TRANSMISSION;

EID: 0033061509     PISSN: 03043959     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0304-3959(98)00191-2     Document Type: Article

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