Sustained hypersensitivity to angiotensin II and its mechanism in mice lacking the subtype-2 (AT2) angiotensin receptor

Proceedings of the National Academy of Sciences of the United States of America

Volumn 96, Issue 11, 1999, Pages 6506-6510

  Siragy, Helmy M ^a   Inagami, Tadashi ^b   Ichiki, Toshihiro ^b   Carey, Robert M ^a,c  

^a UNIVERSITY OF VIRGINIA   (United States)

^b VANDERBILT UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c UNIVERSITY OF VIRGINIA HEALTH SYSTEM   (United States)

Author keywords

Blood pressure; Bradykinin; Sodium

Indexed keywords

ANGIOTENSIN RECEPTOR; RECEPTOR SUBTYPE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BLOOD PRESSURE REGULATION; CONTROLLED STUDY; GENE DELETION; GENE TARGETING; HORMONAL REGULATION; HYPERSENSITIVITY; MOUSE; NONHUMAN; PRESSOR RESPONSE; PRIORITY JOURNAL; RECEPTOR BINDING; RECEPTOR GENE; RENIN ANGIOTENSIN ALDOSTERONE SYSTEM; SODIUM INTAKE;

ANGIOTENSIN II; ANIMALS; BLOOD PRESSURE; BRADYKININ; CYCLIC GMP; DIET, SODIUM-RESTRICTED; HOMOZYGOTE; KIDNEY; MICE; MICE, KNOCKOUT; MICRODIALYSIS; RECEPTOR, ANGIOTENSIN, TYPE 2; RECEPTORS, ANGIOTENSIN; SODIUM; TIME FACTORS; X CHROMOSOME;

ANIMALIA;

EID: 0032990242     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.96.11.6506     Document Type: Article

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