V-Ha-ras gene expression in liver and kidney of transgenic Tg.AC mice following chemically induced tissue injury

Toxicological Sciences

Volumn 50, Issue 1, 1999, Pages 90-97

  Delker, D A ^a   Yano, B L ^a   Gollapudi, B B ^a  

^a DOW CHEMICAL COMPANY   (United States)

Author keywords

Cell proliferation; Chloroform; Gavage; Kidney; Tg.AC mice; Tissue injury; Transgene expression

Indexed keywords

CHLOROFORM; GLOBIN; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR GATA 3; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CARCINOGENESIS; CELL PROLIFERATION; CELL REGENERATION; CHEMICAL INJURY; CONTROLLED STUDY; CYTOTOXICITY; FEMALE; GENE EXPRESSION; GENE INDUCTION; HISTOPATHOLOGY; KIDNEY; LIVER; MALE; MOUSE; NONHUMAN; ONCOGENE; ONCOGENE V HA RAS; TISSUE INJURY; TRANSGENIC MOUSE;

ANIMALS; BROMODEOXYURIDINE; CELL DIVISION; CHLOROFORM; DOSE-RESPONSE RELATIONSHIP, DRUG; FEMALE; GENE EXPRESSION REGULATION; GENES, RAS; KIDNEY; LIVER; MALE; MICE; MICE, TRANSGENIC; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; RNA;

ANIMALIA; MUS MUSCULUS; STAPHYLOCOCCUS PHAGE 3A;

EID: 0032792296     PISSN: 10966080     EISSN: None     Source Type: Journal    
DOI: 10.1093/toxsci/50.1.90     Document Type: Article

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