Mutant RAS selectively promotes sensitivity of myeloid leukemia cells to apoptosis by a protein kinase C-dependent process

Cancer Research

Volumn 58, Issue 9, 1998, Pages 2029-2035

  Gallagher, Angela P ^a   Burnett, Alan K ^a   Bowen, David T ^b   Darley, Richard L ^a  

^a CARDIFF UNIVERSITY   (United Kingdom)

^b UNIVERSITY OF DUNDEE   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

CYTARABINE; DOXORUBICIN; PHORBOL ESTER; PROTEIN KINASE C; RAF PROTEIN; RETINOIC ACID;

ANTIGEN EXPRESSION; APOPTOSIS; ARTICLE; CELL DIFFERENTIATION; CELL PROLIFERATION; DNA DAMAGE; DRUG EFFECT; ENZYME ACTIVATION; ENZYME INHIBITION; GENE MUTATION; HUMAN; HUMAN CELL; MALIGNANT TRANSFORMATION; MYELOID LEUKEMIA; ONCOGENE RAS; PRIORITY JOURNAL; PROTEIN EXPRESSION;

3T3 CELLS; ANIMALS; ANTINEOPLASTIC AGENTS; APOPTOSIS; CELL DIFFERENTIATION; CYTARABINE; DNA PRIMERS; DOSE-RESPONSE RELATIONSHIP, DRUG; DOXORUBICIN; HUMANS; LEUKEMIA, MYELOID; MICE; MUTATION; PROTEIN KINASE C; PROTO-ONCOGENE PROTEINS C-BCL-2; PROTO-ONCOGENE PROTEINS C-RAF; PROTO-ONCOGENE PROTEINS P21(RAS); TRANSFECTION; TRETINOIN; TUMOR CELLS, CULTURED;

EID: 0032080694     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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