Comparative genomic hybridization as a tool to define two distinct chromosome 12-derived amplification units in well-differentiated liposarcomas
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Cytogenetic aberrations in 188 benign and borderline adipose tissue tumors
Mandahl N, Höglund M, Mertens F, et al. Cytogenetic aberrations in 188 benign and borderline adipose tissue tumors. Genes Chromosom Cancer. 1994; 9:207-215.
Gains and losses of DNA sequences in liposarcomas evaluated by comparative genomic hybridization
Szymanska J, Tarkkanen M, Wiklund T, et al. Gains and losses of DNA sequences in liposarcomas evaluated by comparative genomic hybridization. Genes Chromosom Cancer. 1996;15:89-94.
Correlation between clinico-pathologic features and karyotype in lipomatous tumors: A report of 178 cases from the chromosomes and morphology (CHAMP) collaborative study group
Fletcher CDM, Akerman M, Dal Cin P, et al. Correlation between clinico-pathologic features and karyotype in lipomatous tumors: a report of 178 cases from the chromosomes and morphology (CHAMP) collaborative study group. Am J Pathol. 1996;148:623-630.
Translocation t(12;16)(q13;p11) in myxoid liposarcoma and round cell liposarcoma: Molecular and cytogenetic analysis
Knight JC, Renwick P, Dal Cin P, Van Den Berghe H, Fletcher CDM. Translocation t(12;16)(q13;p11) in myxoid liposarcoma and round cell liposarcoma: molecular and cytogenetic analysis. Cancer Res. 1995;55:24-27.
Complex composition and co-amplification of SAS and MDM2 in ring and giant rod marker chromosomes in well-differentiated liposarcoma
Pedeutour F, Suijkerbuijk RF, Forus A, et al. Complex composition and co-amplification of SAS and MDM2 in ring and giant rod marker chromosomes in well-differentiated liposarcoma. Genes Chromosom Cancer. 1994;10:85-94.
HMGI-C rearrangements as the molecular basis for the majority of pulmonary chondroid hamartomas: A survey of 30 tumors
Kazmierczak B, Rosigkeit J, Wanschura S, et al. HMGI-C rearrangements as the molecular basis for the majority of pulmonary chondroid hamartomas: a survey of 30 tumors. Oncogene. 1996;12:515-521.
Translocation breakpoints upstream of the HMGIC gene in uterine leiomyomata suggest dysregulation of this gene by a mechanism different from that in lipomas
Fejzo MS, Ashar HR, Krauter KS, et al. Translocation breakpoints upstream of the HMGIC gene in uterine leiomyomata suggest dysregulation of this gene by a mechanism different from that in lipomas. Genes Chromosom Cancer. 1996; 17:1-6.