Secondary activation of a cation conductance is responsible for NMDA toxicity in acutely isolated hippocampal neurons

Journal of Neuroscience

Volumn 17, Issue 11, 1997, Pages 4032-4036

  Chen, Qiang X ^a   Perkins, Katherine L ^a   Choi, Dennis W ^b   Wong, Robert K S ^a  

^a SUNY DOWNSTATE MEDICAL CENTER   (United States)

^b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

calcium; cell death; excitotoxicity; glutamate; hippocampus; I(pe); neuronal death; neurotoxicity; NMDA; postexposure current; toxicity

Indexed keywords

N METHYL DEXTRO ASPARTIC ACID RECEPTOR;

ARTICLE; CALCIUM CELL LEVEL; CALCIUM TRANSPORT; CELL DEATH; EXTRACELLULAR CALCIUM; HIPPOCAMPUS; NEUROTOXICITY; NONHUMAN; PATCH CLAMP; PHASE CONTRAST MICROSCOPY; PRIORITY JOURNAL; VOLTAGE CLAMP;

EID: 0030987928     PISSN: 02706474     EISSN: None     Source Type: Journal    
DOI: 10.1523/jneurosci.17-11-04032.1997     Document Type: Article

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