Acetylcholinesterase is increased in the brains of transgenic mice expressing the C-terminal fragment (CT100) of the β-amyloid protein precursor of Alzheimer's disease

Journal of Neurochemistry

Volumn 71, Issue 2, 1998, Pages 723-731

  Sberna, Gian ^a   Sáez Valero, Javier ^a   Li, Qiao Xin ^a   Czech, Christian ^b   Beyreuther, Konrad ^b   Masters, Colin L ^a   McLean, Catriona A ^a   Small, David H ^a  

^a UNIVERSITY OF MELBOURNE   (Australia)

^b UNIVERSITY OF HEIDELBERG   (Germany)

Author keywords

Acetylcholinesterase; Alzheimer's disease; Amyloid protein

Indexed keywords

ACETYLCHOLINESTERASE; AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; BETA ACTIN;

ADULT; AGED; ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CARBOXY TERMINAL SEQUENCE; CLINICAL ARTICLE; CONTROLLED STUDY; ENZYME GLYCOSYLATION; FRONTAL CORTEX; HUMAN; HUMAN TISSUE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN EXPRESSION; TRANSGENIC MOUSE;

ANIMALIA; MUS MUSCULUS;

EID: 0031754362     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.1998.71020723.x     Document Type: Article

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