-
2
-
-
0028233494
-
Hydrogen peroxide mediates amyloid β toxicity
-
Behl C., Davis J.B., Lesley R., Schubert D. Hydrogen peroxide mediates amyloid β toxicity. Cell. 77:1994;817-827.
-
(1994)
Cell
, vol.77
, pp. 817-827
-
-
Behl, C.1
Davis, J.B.2
Lesley, R.3
Schubert, D.4
-
3
-
-
0028904180
-
The septohippocampal pathway: Structure and function of a central cholinergic system
-
Dutar P., Bassant M.-H., Senut M.-C., Lamour Y. The septohippocampal pathway: structure and function of a central cholinergic system. Physiol. Rev. 75:1995;393-427.
-
(1995)
Physiol. Rev.
, vol.75
, pp. 393-427
-
-
Dutar, P.1
Bassant, M.-H.2
Senut, M.-C.3
Lamour, Y.4
-
4
-
-
0027220966
-
Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
-
Etcheberrigaray R., Ito E., Oka K., Tofel-Grehl B., Gibson G.E., Alkon D.L. Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease. Proc. Natl. Acad. Sci. USA. 90:1993;8209-8213.
-
(1993)
Proc. Natl. Acad. Sci. USA
, vol.90
, pp. 8209-8213
-
-
Etcheberrigaray, R.1
Ito, E.2
Oka, K.3
Tofel-Grehl, B.4
Gibson, G.E.5
Alkon, D.L.6
-
6
-
-
0021256895
-
Alzheimer's disease: Initial report of the purification and characterization of a novel cerebro-vascular amyloid protein
-
Glenner G.G., Wong C.W. Alzheimer's disease: initial report of the purification and characterization of a novel cerebro-vascular amyloid protein. Biochem. Biophys. Res. Commun. 120:1984;885-890.
-
(1984)
Biochem. Biophys. Res. Commun.
, vol.120
, pp. 885-890
-
-
Glenner, G.G.1
Wong, C.W.2
-
7
-
-
0028938472
-
K-252a promotes survival and choline acetyltransferase activity in striatal and basal forebrain neuronal cultures
-
Glicksman M.A., Forbes M.E., Prantner J.E., Neff N.T. K-252a promotes survival and choline acetyltransferase activity in striatal and basal forebrain neuronal cultures. J. Neurochem. 64:1995;1502-1512.
-
(1995)
J. Neurochem.
, vol.64
, pp. 1502-1512
-
-
Glicksman, M.A.1
Forbes, M.E.2
Prantner, J.E.3
Neff, N.T.4
-
8
-
-
0031025045
-
Nontoxic amyloid β peptide 1-42 suppresses acetylcholine synthesis
-
Hoshi M., Takashima A., Murayama M., Yasutake K., Yoshida N., Ishiguro K., Hoshino T., Imahori K. Nontoxic amyloid β peptide 1-42 suppresses acetylcholine synthesis. J. Biol. Chem. 272:1997;2038-2041.
-
(1997)
J. Biol. Chem.
, vol.272
, pp. 2038-2041
-
-
Hoshi, M.1
Takashima, A.2
Murayama, M.3
Yasutake, K.4
Yoshida, N.5
Ishiguro, K.6
Hoshino, T.7
Imahori, K.8
-
10
-
-
0029996479
-
β-Amyloid-related peptides inhibit potassium-evoked acetylcholine release from rat hippocampal slices
-
Kar S., Seto D., Gaudreau P., Quirion R. β-Amyloid-related peptides inhibit potassium-evoked acetylcholine release from rat hippocampal slices. J. Neurosci. 16:1996;1034-1040.
-
(1996)
J. Neurosci.
, vol.16
, pp. 1034-1040
-
-
Kar, S.1
Seto, D.2
Gaudreau, P.3
Quirion, R.4
-
11
-
-
0029900084
-
Amyloid β-peptide disrupts carbachol-induced muscarinic cholinergic signal transduction in cortical neurons
-
Kelly J.F., Furukawa K., Barger S.W., Rengen M.R., Mark R.J., Blanc E.M., Roth G.S., Mattson M.P. Amyloid β-peptide disrupts carbachol-induced muscarinic cholinergic signal transduction in cortical neurons. Proc. Natl. Acad. Sci. USA. 93:1996;6753-6758.
-
(1996)
Proc. Natl. Acad. Sci. USA
, vol.93
, pp. 6753-6758
-
-
Kelly, J.F.1
Furukawa, K.2
Barger, S.W.3
Rengen, M.R.4
Mark, R.J.5
Blanc, E.M.6
Roth, G.S.7
Mattson, M.P.8
-
12
-
-
0029671451
-
Water-soluble Aβ (N-40, N-42) oligomers in normal and Alzheimer disease brains
-
Kuo Y.-M., Emmerling M.R., Vigo-Pelfrey C., Kasunic T.C., Kirkpatrick J.B., Murdoch G.H., Ball M.J., Roher A.E. Water-soluble Aβ (N-40, N-42) oligomers in normal and Alzheimer disease brains. J. Biol. Chem. 271:1996;4077-4081.
-
(1996)
J. Biol. Chem.
, vol.271
, pp. 4077-4081
-
-
Kuo, Y.-M.1
Emmerling, M.R.2
Vigo-Pelfrey, C.3
Kasunic, T.C.4
Kirkpatrick, J.B.5
Murdoch, G.H.6
Ball, M.J.7
Roher, A.E.8
-
13
-
-
0028978360
-
Physiological levels of β-amyloid increase tyrosine phosphorylation and cytosolic calcium
-
Luo Y.Q., Hirashima N., Li Y.H., Alkon D.L., Sunderland T., Etcheberrigaray R., Wolozin B. Physiological levels of β-amyloid increase tyrosine phosphorylation and cytosolic calcium. Brain Res. 681:1995;65-74.
-
(1995)
Brain Res.
, vol.681
, pp. 65-74
-
-
Luo, Y.Q.1
Hirashima, N.2
Li, Y.H.3
Alkon, D.L.4
Sunderland, T.5
Etcheberrigaray, R.6
Wolozin, B.7
-
14
-
-
0029837577
-
Physiologic levels of β-amyloid activate phosphatidylinositol 3-kinase with the involvement of tyrosine phosphorylation
-
Luo Y., Sunderland T., Wolozin B. Physiologic levels of β-amyloid activate phosphatidylinositol 3-kinase with the involvement of tyrosine phosphorylation. J. Neurochem. 67:1996;978-987.
-
(1996)
J. Neurochem.
, vol.67
, pp. 978-987
-
-
Luo, Y.1
Sunderland, T.2
Wolozin, B.3
-
15
-
-
0030592575
-
Physiological levels of β-amyloid peptide promote PC12 cell proliferation
-
Luo Y., Sunderland T., Roth G.S., Wolozin B. Physiological levels of β-amyloid peptide promote PC12 cell proliferation. Neurosci. Lett. 217:1996;125-128.
-
(1996)
Neurosci. Lett.
, vol.217
, pp. 125-128
-
-
Luo, Y.1
Sunderland, T.2
Roth, G.S.3
Wolozin, B.4
-
16
-
-
0000131472
-
Calcium homeostasis and free radical metabolism as convergence points in the pathophysiology of dementia
-
In W. Wasco and R.E. Tanzi (Eds.), Humana Press, Totowa, NJ, 312 pp.
-
Mattson, M.P., Furukawa, K., Bruce, A.J., Mark, R.J. and Blanc, E., Calcium homeostasis and free radical metabolism as convergence points in the pathophysiology of dementia. In W. Wasco and R.E. Tanzi (Eds.), Molecular Mechanisms of Dementia, Humana Press, Totowa, NJ, 1996, 312 pp.
-
(1996)
Molecular Mechanisms of Dementia
-
-
Mattson, M.P.1
Furukawa, K.2
Bruce, A.J.3
Mark, R.J.4
Blanc, E.5
-
17
-
-
0029043420
-
All-trans- And 9-cis-retinoic acid enhance the cholinergic properties of a murine septal cell line: Evidence that the effects are mediated by activation of retinoic acid receptor-α
-
Pedersen W.A., Berse B., Schuler U., Wainer B.H., Blusztajn J.K. All-trans- and 9-cis-retinoic acid enhance the cholinergic properties of a murine septal cell line: evidence that the effects are mediated by activation of retinoic acid receptor-α J. Neurochem. 65:1995;50-58.
-
(1995)
J. Neurochem.
, vol.65
, pp. 50-58
-
-
Pedersen, W.A.1
Berse, B.2
Schuler, U.3
Wainer, B.H.4
Blusztajn, J.K.5
-
18
-
-
0029795254
-
Amyloid-β protein reduces acetylcholine synthesis in a cell line derived from cholinergic neurons of the basal forebrain
-
Pedersen W.A., Kloczewiak M.A., Blusztajn J.K. Amyloid-β protein reduces acetylcholine synthesis in a cell line derived from cholinergic neurons of the basal forebrain. Proc. Natl. Acad. Sci. USA. 93:1996;8068-8071.
-
(1996)
Proc. Natl. Acad. Sci. USA
, vol.93
, pp. 8068-8071
-
-
Pedersen, W.A.1
Kloczewiak, M.A.2
Blusztajn, J.K.3
-
19
-
-
0028267440
-
Normal and abnormal biology of the β-amyloid precursor protein
-
Selkoe D.J. Normal and abnormal biology of the β-amyloid precursor protein. Annu. Rev. Neurosci. 17:1994;489-517.
-
(1994)
Annu. Rev. Neurosci.
, vol.17
, pp. 489-517
-
-
Selkoe, D.J.1
-
20
-
-
0026646605
-
Isolation and quantification of soluble Alzheimer's β-peptide from biological fluids
-
Seubert P., Vigo-Pelfrey C., Esch F., Lee M., Dovey H., Davis D., Sinha S., Schlossmacher M., Whaley J., Swindlehurst C., McCormack R., Wolfert R., Selkoe D.J., Lieberburg I., Schenk D. Isolation and quantification of soluble Alzheimer's β-peptide from biological fluids. Nature. 359:1992;325-327.
-
(1992)
Nature
, vol.359
, pp. 325-327
-
-
Seubert, P.1
Vigo-Pelfrey, C.2
Esch, F.3
Lee, M.4
Dovey, H.5
Davis, D.6
Sinha, S.7
Schlossmacher, M.8
Whaley, J.9
Swindlehurst, C.10
McCormack, R.11
Wolfert, R.12
Selkoe, D.J.13
Lieberburg, I.14
Schenk, D.15
-
21
-
-
0029120145
-
Dissociation of nuclear and cytoplasmic cell cycle progression by drugs employed in cell synchronization
-
Urbani L., Sherwood S.W., Schimke R.T. Dissociation of nuclear and cytoplasmic cell cycle progression by drugs employed in cell synchronization. Exp. Cell Res. 219:1995;159-168.
-
(1995)
Exp. Cell Res.
, vol.219
, pp. 159-168
-
-
Urbani, L.1
Sherwood, S.W.2
Schimke, R.T.3
-
22
-
-
0028174684
-
Concentrations of amyloid-β protein in cerebrospinal fluid increase with age in patients free from neurodegenerative disease
-
van Gool W.A., Schenk D.B., Bolhuis P.A. Concentrations of amyloid-β protein in cerebrospinal fluid increase with age in patients free from neurodegenerative disease. Neurosci. Lett. 172:1994;122-124.
-
(1994)
Neurosci. Lett.
, vol.172
, pp. 122-124
-
-
Van Gool, W.A.1
Schenk, D.B.2
Bolhuis, P.A.3
-
23
-
-
0029896354
-
Mechanisms of neuronal degeneration in Alzheimer's disease
-
Yankner B.A. Mechanisms of neuronal degeneration in Alzheimer's disease. Neuron. 16:1996;921-932.
-
(1996)
Neuron
, vol.16
, pp. 921-932
-
-
Yankner, B.A.1
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