DNA-dependent protein kinase is not required for accumulation of p53 or cell cycle arrest after DNA damage

Cancer Research

Volumn 57, Issue 1, 1997, Pages 68-74

  Rathmell, W Kiniryn ^a   Kaufmann, William K ^b   Hurt, John C ^b   Byrd, Laura L ^b   Chu, Gilbert ^a  

^a STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF NORTH CAROLINA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN KINASE;

ANIMAL CELL; ARTICLE; CATALYSIS; CELL CULTURE; CELL CYCLE; COMBINED IMMUNODEFICIENCY; DNA DAMAGE; DNA DETERMINATION; DNA SEQUENCE; DNA TRANSCRIPTION; GENE ACTIVATION; GENETIC TRANSDUCTION; IONIZING RADIATION; MITOSIS INHIBITION; NONHUMAN; NORTHERN BLOTTING; PRIORITY JOURNAL; SEQUENCE HOMOLOGY; SIGNAL TRANSDUCTION; ULTRAVIOLET RADIATION;

ANIMALS; ANIMALS, NEWBORN; CCAAT-ENHANCER-BINDING PROTEINS; CRICETINAE; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; DNA; DNA DAMAGE; DNA-ACTIVATED PROTEIN KINASE; DNA-BINDING PROTEINS; FIBROBLASTS; G1 PHASE; G2 PHASE; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MICE; MICE, INBRED BALB C; MICE, SCID; PROTEIN-SERINE-THREONINE KINASES; PROTEINS; TRANSCRIPTION FACTOR CHOP; TRANSCRIPTION FACTORS; TRANSCRIPTION, GENETIC; TUMOR SUPPRESSOR PROTEIN P53;

EID: 0031025079     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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