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Volumn 3, Issue 7, 1997, Pages 723-725

Serpents on the road to dementia and death

Author keywords

[No Author keywords available]

Indexed keywords

ALZHEIMER DISEASE; AMYLOIDOSIS; DEATH; DEMENTIA; GENE MUTATION; HUMAN; NERVE DEGENERATION; PRIORITY JOURNAL; SHORT SURVEY;

EID: 0030791479     PISSN: 10788956     EISSN: None     Source Type: Journal    
DOI: 10.1038/nm0797-723     Document Type: Short Survey
Times cited : (14)

References (12)
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  • 3
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    • Presenilin 1 is required for Notch 1 and DII 1 expression in the paraxial mesoderm
    • Wong, P.C. et al. Presenilin 1 is required for Notch 1 and DII 1 expression in the paraxial mesoderm. Nature 387, 288-292 (1997)
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    • Wong, P.C.1
  • 4
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    • Skeletal and CNS defects in Presenilin-1-deficient mice
    • Shen, J. et al. Skeletal and CNS defects in Presenilin-1-deficient mice. Cell 89, 629-639 (1997).
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    • Shen, J.1
  • 5
    • 16044362074 scopus 로고    scopus 로고
    • Notch 3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia
    • Joutel, A. et al. Notch 3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia. Nature 383, 707-710 (1996).
    • (1996) Nature , vol.383 , pp. 707-710
    • Joutel, A.1
  • 6
    • 16044366039 scopus 로고    scopus 로고
    • Increased amyloid-β342(43) in brains of mice expressing mutant presenilin 1
    • Duff, K. et al. Increased amyloid-β342(43) in brains of mice expressing mutant presenilin 1. Nature 383, 710-713 (1996).
    • (1996) Nature , vol.383 , pp. 710-713
    • Duff, K.1
  • 7
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    • Scheuner, D. et al. Secreted amyloid-β protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease. Nature Med. 2, 864-870 (1996).
    • (1996) Nature Med. , vol.2 , pp. 864-870
    • Scheuner, D.1
  • 8
    • 0031054505 scopus 로고    scopus 로고
    • Formation of stable complexes between two Alzheimer's disease gene products: Presenilin 2 and β-amyloid precursor protein
    • Weidemann, A. et al. Formation of stable complexes between two Alzheimer's disease gene products: Presenilin 2 and β-amyloid precursor protein. Nature Med. 3, 328-332 (1997).
    • (1997) Nature Med. , vol.3 , pp. 328-332
    • Weidemann, A.1
  • 9
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    • Neuronal sorting and processing of amyloid precursor protein: Implications for Alzheimer's disease
    • Tienari, P. J. et al. Neuronal sorting and processing of amyloid precursor protein: implications for Alzheimer's disease. Cold Spring Harbor Symp. Quant. Biol. 56, 575-585 (1996).
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  • 10
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    • Impaired learning and LTP in mice expressing the carboxy terminus of the Alzheimer amyloid precursor protein
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  • 11
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    • Mutations in mitochondrial coytochrome c oxidase genes segregate with late-onset Alzheimer's disease
    • Davis, R. E. et al. Mutations in mitochondrial coytochrome c oxidase genes segregate with late-onset Alzheimer's disease. Proc. Natl. Acad. Sci. USA 94, 4526-4531 (1997).
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  • 12
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    • Amyloid deposition is delayed in mice with targeted deletion of the serum amyloid P component gene
    • in the press
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.