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1
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0027027615
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Multifactorial analysis of renal transplants reported to the United Network for Organ Sharing Registry
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P. Terasaki, Cecka M. Los Angeles: UCLA Tissue Typing Laboratory
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Gjertson DW. Multifactorial analysis of renal transplants reported to the United Network for Organ Sharing Registry. Terasaki P, Cecka M. Clinical Transplants 1992. 1993;299-317 UCLA Tissue Typing Laboratory, Los Angeles.
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Gjertson, D.W.1
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2
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0027326797
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International standardization of criteria for the histologic diagnosis of renal allograft rejection: The Banff working classification of kidney transplant pathology
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Solez K, Axelsen RA, Benediktsson H, Burdick JF, Cohen AH, Colvin RB, Croker BP, Droz D, Dunnill MS, Halloran PF, et al. International standardization of criteria for the histologic diagnosis of renal allograft rejection: the Banff working classification of kidney transplant pathology. Kidney Int. 44:1993;411-422.
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Solez, K.1
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Solez, K.1
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5
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0000508256
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The impact of ischemia/reperfusion injury on specific and non-specific, early and late chronic events after organ transplantation
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of special interest. An excellent overview of chronic allograft rejection with a clinical perspective. The primary focus is on the vascular biology of ischernia/reperfusion injury and its effects on later events leading to chronic graft failure
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Land W, Messmer K. The impact of ischemia/reperfusion injury on specific and non-specific, early and late chronic events after organ transplantation. of special interest Transplant Rev. 10:1996;236-253 An excellent overview of chronic allograft rejection with a clinical perspective. The primary focus is on the vascular biology of ischernia/reperfusion injury and its effects on later events leading to chronic graft failure.
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Transplant Rev
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Land, W.1
Messmer, K.2
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6
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Orosz CG, Bergese SD, Wakely E, Xia D, Gordillo GM, Van Buskirk AM. Acute versus chronic graft rejection: related manifestations of allosensitization in graft recipients. Transplant Rev. 11:1997;38-50.
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Orosz, C.G.1
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Van Buskirk, A.M.6
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7
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Molecular mechanisms of chronic renal allograft rejection
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Lemstrom K, Koskinen P, Hayry P. Molecular mechanisms of chronic renal allograft rejection. Kidney Int. 48:1995;S2-S10. (suppl 52).
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Lemstrom, K.1
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8
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Azuma H, Tilney NL. Immune and nonimmune mechanisms of chronic rejection of kidney allografts. J Heart Lung Transplant. 14:1995;S136-S142.
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Orosz CG, Sedmak DD. Concerns regarding the current paradigm for chronic allograft rejection. Trans Immunol. 1997;. in press.
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Orosz, C.G.1
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Pathophysiology of vascular wall changes in chronic allograft rejection
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Hayry P, Mennander A, Raisanen-Sokolowski A, Ustinov J, Lemstrom K, Aho P, Yilmaz S, Lautenschlager I, Paavonen T. Pathophysiology of vascular wall changes in chronic allograft rejection. Transplant Rev. 7:1993;1-20.
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Hayry, P.1
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Kato, Y.1
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Yilmaz S, Paavonen T, Hayry P. Chronic rejection of rat renal allografts. II. The impact of prolonged ischemia time on transplant histology. Transplantation. 53:1992;823-827.
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0028301760
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Russell PS, Chase CM, Winn HJ, Colvin RB. Coronary atherosclerosis in transplanted mouse hearts. II. Importance of humoral immunity. J Immunol. 152:1994;5135-5141.
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Russell, P.S.1
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15
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0028917962
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Upregulation of cytokines associated with macrophage activation in the Lewis-to-F344 rat transplantation model of chronic cardiac rejection
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Russell ME, Wallace AF, Hancock WW, Sayegh MH, Adams DH, Sibinga NES, Wyner LR, Kamovsky MJ. Upregulation of cytokines associated with macrophage activation in the Lewis-to-F344 rat transplantation model of chronic cardiac rejection. Transplantation. 59:1995;572-578.
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Russell, M.E.1
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16
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Experimental graft arteriosclerosis. II. Immunocytochemical analysis of lesion development
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Adams DH, Wyner LR, Karnovsky MJ. Experimental graft arteriosclerosis. II. Immunocytochemical analysis of lesion development. Transplantation. 56:1993;794-799.
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Adams, D.H.1
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Karnovsky, M.J.3
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0028277677
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Coronary atherosclerosis in transplanted mouse hearts. 1. Time course and immunogenetic and immunopathologicalal considerations
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Russell PS, Chase CM, Winn HJ, Colvin RB. Coronary atherosclerosis in transplanted mouse hearts. 1. Time course and immunogenetic and immunopathologicalal considerations. Am J Pathol. 144:1994;260-270.
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Russell, P.S.1
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Winn, H.J.3
Colvin, R.B.4
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18
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0029115886
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Sequential immunological targeting of chronic experimental arterial allograft
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Plissonnier D, Nochy D, Poncet P, Mandet C, Hinglais N, Bariety J, Michel JB. Sequential immunological targeting of chronic experimental arterial allograft. Transplantation. 60:1995;414-424.
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Plissonnier, D.1
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Mandet, C.4
Hinglais, N.5
Bariety, J.6
Michel, J.B.7
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19
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0029966296
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Immunologic basis of transplant-associated arteriosclerosis
-
of outstanding interest. A revealing series of studies conducted with mutant mouse strains that demonstrates the immunological conditions (i.e. requirements for macrophages, T cell subsets, etc.) necessary for rapid neointimal formation in carotid grafts.
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Shi CW, Lee W-S, He Q, Zhang D, Fletcher DL Jr, Newell JB, Haber E. Immunologic basis of transplant-associated arteriosclerosis. of outstanding interest Proc Natl Acad Sci USA. 93:1996;4051-4056 A revealing series of studies conducted with mutant mouse strains that demonstrates the immunological conditions (i.e. requirements for macrophages, T cell subsets, etc.) necessary for rapid neointimal formation in carotid grafts.
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Proc Natl Acad Sci USA
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Shi, C.W.1
Lee W-S2
He, Q.3
Zhang, D.4
Fletcher D.L., Jr.5
Newell, J.B.6
Haber, E.7
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20
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-
0029835479
-
Networked alloimmunity: A brief reexamination of some basic concepts in transplantation immunobiology
-
of special interest. A view of immunobiology that accepts complexity as an indispensable element of immune responses. This view suggests that many of the current experimental approaches may be fundamentally flawed because they either ignore complexity as a formative element, or they limit it so severely that the experimental outcomes lose their biological relevance
-
Orosz CG. Networked alloimmunity: a brief reexamination of some basic concepts in transplantation immunobiology. of special interest J Heart Lung Transplant. 15:1996;1063-1068 A view of immunobiology that accepts complexity as an indispensable element of immune responses. This view suggests that many of the current experimental approaches may be fundamentally flawed because they either ignore complexity as a formative element, or they limit it so severely that the experimental outcomes lose their biological relevance.
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(1996)
J Heart Lung Transplant
, vol.15
, pp. 1063-1068
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Orosz, C.G.1
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21
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0031019745
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Isolation of putative progenitor endothelial cells for anglogenesis
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Asahara T, Murohara T, Sullivan A, Silver M, Van der Zee R, Li T, Witzenbichler B, Schatteman G, Isner JM. Isolation of putative progenitor endothelial cells for anglogenesis. Science. 275:1997;964-967.
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Asahara, T.1
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Silver, M.4
Van Der Zee, R.5
Li, T.6
Witzenbichler, B.7
Schatteman, G.8
Isner, J.M.9
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22
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0028535872
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Bucala R, Spiegel LA, Chesney J, Hogan M, Cerami A. Circulating fibrocytes define a new leukocyte subpopulation that mediates tissue repair. Mol Med. 1:1994;71-81.
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Bucala, R.1
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Chesney, J.3
Hogan, M.4
Cerami, A.5
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23
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0028802192
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Evidence that graft coronary arteriosclerosis begins in the early phase after transplantation and progresses without chronic immunoreaction: Histopathologicalal analysis using a retransplantation model
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Izutani H, Miyagawa S, Shirakura R, Matsumiya G, Nakata S, Shimazaki Y, Matsuda H. Evidence that graft coronary arteriosclerosis begins in the early phase after transplantation and progresses without chronic immunoreaction: histopathologicalal analysis using a retransplantation model. Transplantation. 60:1995;1073-1079.
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Izutani, H.1
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Matsuda, H.7
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24
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0029902010
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Retransplantation reverses mononuclear infiltration but not myointimal proliferation in a rat model of chronic cardiac allograft rejection
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of special interest. These studies highlight the influence of peri-transplant trauma on vascular remodeling by demonstrating that retransplantation of cardiac isografts promotes the development of transplant vasculopathy that is as rapid and severe as that observed in the retransplantation of cardiac allografts
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Schmid C, Heemann U, Tilney NL. Retransplantation reverses mononuclear infiltration but not myointimal proliferation in a rat model of chronic cardiac allograft rejection. of special interest Transplantation. 61:1996;1695-1699 These studies highlight the influence of peri-transplant trauma on vascular remodeling by demonstrating that retransplantation of cardiac isografts promotes the development of transplant vasculopathy that is as rapid and severe as that observed in the retransplantation of cardiac allografts.
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Transplantation
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Schmid, C.1
Heemann, U.2
Tilney, N.L.3
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Immunol Today
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Wick, G.1
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0028358704
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Ferguson R. Acute rejection episodes - best predictor of long-term primary cadaveric renal transplant survival. Clin Transplant. 8:1994;328-331.
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The impact of an acute rejection episode on long-term renal allograft survival (t1/2)
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Matas AJ, Gillingham KJ, Payne WD, Najarian JS. The impact of an acute rejection episode on long-term renal allograft survival (t1/2). Transplantation. 57:1994;857-859.
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Matas, A.J.1
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0028043866
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Tullius SG, Hancock WW, Heemann UW. Long-term kidney isografts develop functional and morphological changes which mimic those of chronic allograft rejection. Ann Surg. 220:1994;425-432.
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Tullius, S.G.1
Hancock, W.W.2
Heemann, U.W.3
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Davies MG, Hagen PO. Structural and functional consequences of bypass grafting with autologous vein. Cryobiology. 31:1994;63-70.
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0029083026
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Phenotypic modulation of smooth muscle cells during the formation of neointimal thickenings in the rat carotid artery after balloon injury: An electron-microscopic and stereological study
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Thyberg J, Blomgren K, Hedin U, Dryjski M. Phenotypic modulation of smooth muscle cells during the formation of neointimal thickenings in the rat carotid artery after balloon injury: An electron-microscopic and stereological study. Cell Tissue Res. 281:1995;421-433.
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0029840473
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Grider, L.7
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32
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0031046924
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Nephron mass modulates the hemodynamic, cellular, and molecular response of the rat renal allograft
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of special interest. A definitive work demonstrating that chronic physiological insufficiency caused by reduced blood flow in rat kidneys can stimulate profound changes in organ function and architecture, including the development of chronic rejection-like histopathology
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Azuma H, Nadeau K, Mackenzie HS, Brenner BM, Tilney NL. Nephron mass modulates the hemodynamic, cellular, and molecular response of the rat renal allograft. of special interest Transplantation. 63:1997;519-528 A definitive work demonstrating that chronic physiological insufficiency caused by reduced blood flow in rat kidneys can stimulate profound changes in organ function and architecture, including the development of chronic rejection-like histopathology.
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Azuma, H.1
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0027425652
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Zhao X-M, Yeoh T-K, Hiebert M, Frist WH, Miller GG. The expression of acidic fibroblast growth factor (heparin-binding growth factor-1) and cytokine genes in human cardiac allografts and T cells. Transplantation. 56:1993;1177-1182.
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Zhao X-M1
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Frist, W.H.4
Miller, G.G.5
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34
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T lymphocytes that infiltrate tumors and atherosclerotic plaques produce heparin-binding epidermal growth factor-like growth factor and basic fibroblast growth factor: A potential pathological role
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Peoples GE, Blotnick S, Takahashi K, Freeman MR, Klagsbrun M, Eberlein TJ. T lymphocytes that infiltrate tumors and atherosclerotic plaques produce heparin-binding epidermal growth factor-like growth factor and basic fibroblast growth factor: a potential pathological role. Proc Natl Acad Sci USA. 92:1995;6547-6551.
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Regulatory T cell clones induced by oral tolerance: Suppression of autoimmune encephalomyelitis
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Chen, Y.1
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