Chronic cocaine administration increases tyrosine hydroxylase activity in the ventral tegmental area through glutaminergic- and dopaminergic D2-receptor mechanisms

Neuroscience Letters

Volumn 217, Issue 2-3, 1996, Pages 73-76

  Masserano, Joseph M ^a   Baker, Ivory ^a   Natsukari, Naoki ^a   Wyatt, Richard Jed ^a  

^a US National Institutes of Health   (United States)

Author keywords

Clozapine; Cocaine; Haloperidol; MK 801; Tyrosine hydroxylase; Ventral tegmental area

Indexed keywords

8 CHLORO 2,3,4,5 TETRAHYDRO 3 METHYL 5 PHENYL 1H 3 BENZAZEPIN 7 OL HYDROGEN MALEATE; CLOZAPINE; COCAINE; DIZOCILPINE; DOPAMINE 2 RECEPTOR; DOPAMINE RECEPTOR BLOCKING AGENT; HALOPERIDOL; TYROSINE 3 MONOOXYGENASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVITY; INTRAPERITONEAL DRUG ADMINISTRATION; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; VENTRAL TEGMENTUM;

EID: 0030592569     PISSN: 03043940     EISSN: None     Source Type: Journal    
DOI: 10.1016/0304-3940(96)13047-0     Document Type: Article

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