Neither gastric topological distribution nor principle virulence genes of Helicobacter pylori contributes to clinical outcomes

World Journal of Gastroenterology

Volumn 10, Issue 22, 2004, Pages 3274-3277

  Ho, Yan Wing ^a   Ho, Khek Yu ^a   Ascencio, Felipe ^b   Ho, Bow ^a  

^a NATIONAL UNIVERSITY OF SINGAPORE   (Singapore)

^b CENTRO DE INVESTIGACIONES BIOLÓGICAS DEL NOROESTE   (Mexico)

Author keywords

[No Author keywords available]

Indexed keywords

BACTERIAL DNA; CAGA PROTEIN; VACUOLATING TOXIN; VIRULENCE FACTOR;

ADULT; AGED; ARTICLE; ASIAN; BACTERIAL COLONIZATION; BACTERIAL GENE; BACTERIAL STRAIN; BACTERIAL VIRULENCE; BACTERIUM DETECTION; BACTERIUM ISOLATE; CARDIA; CONTROLLED STUDY; DIAGNOSTIC PROCEDURE; DISEASE ASSOCIATION; DNA FINGERPRINTING; DNA POLYMORPHISM; DYSPEPSIA; ENDOSCOPIC BIOPSY; ETHNOLOGY; FEMALE; GASTROESOPHAGEAL REFLUX; GASTROINTESTINAL DISEASE; GENE AMPLIFICATION; GENOTYPE; HEALTH STATUS; HELICOBACTER PYLORI; HUMAN; HUMAN TISSUE; INFORMED CONSENT; MAJOR CLINICAL STUDY; MALE; NONHUMAN; OUTCOMES RESEARCH; PEPTIC ULCER; POLYMERASE CHAIN REACTION; POPULATION RESEARCH; PREVALENCE; SINGAPORE; STOMACH ANTRUM; STOMACH BIOPSY; SYMPTOMATOLOGY; TISSUE DISTRIBUTION; UPPER GASTROINTESTINAL TRACT;

EID: 9744273885     PISSN: 10079327     EISSN: None     Source Type: Journal    
DOI: 10.3748/wjg.v10.i22.3274     Document Type: Article

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